Why Does My Left Side Hurt After Drinking Alcohol?

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Can your left kidney hurt from drinking?

The lowdown – Your kidneys constantly work to remove toxins from your blood and balance the amount of water in your body. They work overtime when you’re drinking alcohol. Binge drinking, in particular, stresses your kidneys and can lead to problems that result in mild to severe kidney pain.

Kidney pain can be caused by many factors, including dehydration, kidney stones, and urinary tract infections. If you believe you are having kidney pain after drinking alcohol, schedule an appointment with your healthcare provider. Seeking professional medical care sooner than later can help you get an accurate diagnosis of what is causing your kidney pain.

They will assist you in introducing an individualized treatment plan that will relieve your symptoms and preserve the health of your kidneys.

Can alcohol cause left rib pain?

This article is medically reviewed by Consultant Psychiatrist Prof. Jonathan Chick, MA (Cantab), MPhil, MBChB, DSc, FRCPsych, FRCPE. Rehab Costs & Options for Alcohol | Drugs | Other addictions Have you ever had an ache in your chest after a night of heavy drinking, perhaps over the holidays? Or maybe you even felt it a few hours after a binge-drinking session.

1. If this sounds familiar, you may have a serious problem.
2. Although not a defined condition, some doctors have labelled the irregular heartbeat, breathlessness, or chest pain after alcohol consumption as “holiday heart syndrome”.
3. The British Medical Journal reported Sunday night into Monday morning is the commonest time of the week for heart deaths in the UK and linked this to weekend binge drinking (1),

Pain in the chest can indicate a variety of problems so, before diagnosing chest pain as due to the heart, doctors will of course also consider whether it might be arising in the oesophagus (‘gullet’) and stomach – which might also be related to alcohol from alcohol-irritation – or strain in one of the rib muscles.

How do I know if my liver hurts from alcohol?

Causes – Alcoholic liver disease occurs after years of heavy drinking. Over time, scarring and cirrhosis can occur. Cirrhosis is the final phase of alcoholic liver disease. Alcoholic liver disease does not occur in all heavy drinkers. The chances of getting liver disease go up the longer you have been drinking and more alcohol you consume.

1. You do not have to get drunk for the disease to happen.
2. The disease is common in people between 40 and 50 years of age.
3. Men are more likely to have this problem.
4. However, women may develop the disease after less exposure to alcohol than men.
5. Some people may have an inherited risk for the disease.
6. Long-term alcohol abuse can lead to dangerous damage called alcoholic liver disease.

Let’s talk today about alcoholic liver disease. Alcoholic liver disease usually occurs after years of drinking too much. The longer you’ve abused alcohol, and the more alcohol you’ve consumed, the greater likelihood you will develop liver disease. Alcohol may cause swelling and inflammation in your liver, or something called hepatitis.

Over time, this can lead to scarring and cirrhosis of the liver, which is the final phase of alcoholic liver disease. The damage caused by cirrhosis is unfortunately irreversible. To determine if you have alcoholic liver disease your doctor will probably test your blood, take a biopsy of the liver, and do a liver function test.

You should also have other tests to rule out other diseases that could be causing your symptoms. Your symptoms may vary depending upon the severity of your disease. Usually, symptoms are worse after a recent period of heavy drinking. In fact, you may not even have symptoms until the disease is pretty advanced.

Generally, symptoms of alcoholic liver disease include abdominal pain and tenderness, dry mouth and increased thirst, fatigue, jaundice (which is yellowing of the skin), loss of appetite, and nausea. Your skin may look abnormally dark or light. Your feet or hands may look red. You may notice small, red, spider-like blood vessels on your skin.

You may have abnormal bleeding. Your stools might be dark, bloody, black, or tarry. You may have frequent nosebleeds or bleeding gums. You may vomit blood or material that looks like coffee grounds. Alcoholic liver disease also can affect your brain and nervous system.

Symptoms include agitation, changing mood, confusion, and pain, numbness, or a tingling sensation in your arms or legs. The most important part of treatment is to stop drinking alcohol completely. If you don’t have liver cirrhosis yet, your liver can actually heal itself, that is, if you stop drinking alcohol.

You may need an alcohol rehabilitation program or counseling to break free from alcohol. Vitamins, especially B-complex vitamins and folic acid, can help reverse malnutrition. If cirrhosis develops, you will need to manage the problems it can cause. It may even lead to needing a liver transplant.

Is it normal for organs to hurt after drinking?

If you’ve experienced pain with drinking alcohol, what could it mean? Pain after drinking alcohol has been associated with Hodgkin lymphoma. In general, though, there are usually other reasons for the pain. Drinking too much can cause the familiar hangover and the general feeling of being unwell.

It also may cause irritation or pain in the area over the stomach, esophagus, pancreas, gallbladder, and liver. There is a long list of disorders and diseases linked to excessive alcohol use. This article discusses these conditions, some of which may be quite serious. It will help you to be familiar with symptoms of these illnesses and the causes of pain linked to drinking alcohol.

Verywell / Elise Degarmo

What organ does alcohol damage first?

Multi-Organ Alcohol-Related Damage: Mechanisms and Treatment Alcohol consumption causes damage to various organs and systems. Liver is a primary target for the detrimental effects of alcohol since this substance of abuse is mainly metabolized by liver cells, which express high levels of two major alcohol oxidizing enzymes, alcohol dehydrogenase and CYP2E1.

However, other organs, including brain, gut, pancreas, lungs and the immune system are also affected by alcohol. Alcohol may also serve to intensify the progression of viral infections, autoimmune diseases and cancer. Common mechanisms of alcohol-related organ injury include increases in oxidative stress, methylation impairments, aberrant posttranslational modifications of proteins, dysregulation in lipid metabolism and signal transduction pathways, all of which ultimately affect cell survival and function.

The Special Issue on “Multi-Organ Alcohol-Related Damage: Mechanisms and Treatment” presents 17 review and 9 original articles which cover diverse topics related to the pathogenesis of organ dysfunction by alcohol exposure. Here, we provide a short summary of all the articles published in this Special Issue.

Several articles address common mechanisms of multi-organ alcohol-induced organ damage. In this regard, the paper by Natarajan et al. discusses the role of microRNAs in the pathogenesis of alcohol-induced pancreatitis, liver damage, cardiomyopathy, muscle damage, intestinal epithelial barrier dysfunction and brain damage, particularly the altered hippocampus structure and function and neuronal loss.

The review by Anji and Kumari focusses on the effect of alcohol on different RNA-binding proteins and their possible contribution to alcohol-related disorders. The authors present a thoughtful deliberation on the role of these proteins in the development of neurological diseases and cancer as well as the conventional methods and newer techniques that are employed to identify RNA-binding proteins.

1. The review by Steiner et al.
2. Discusses the glucose metabolic effects of alcohol in the muscle, liver and adipose tissue under basal post-absorptive conditions and in response to insulin stimulation.
3. They present evidence that alcohol intake is related to the development and/or exacerbation of type 2 diabetes and demonstrate the necessity of employing a multi-systems approach since both alcohol and diabetes affect multiple targets within the body.

Ji presents the recent advances in alcohol-induced organelle stress, unfolded protein responses and organ damage. His review describes the alcohol-altered proteostasis, endoplasmic reticulum stress response as well as discusses new concepts on alcohol-induced mitochondrial, Golgi and lysosomal stress responses in the pathogenesis of tissue injury.

• Since liver is a primary site of ethanol metabolism and liver cells are susceptible to alcohol-induced damage, several articles in this special issue are on the effects of alcohol in promoting liver injury.
• Among them is the study by Shukla et al.
• That focuses on the epigenetic changes induced by acute and chronic ethanol consumption.

They clearly demonstrate that ethanol ingestion elicit characteristic profiles of histone modifications, metabolic alterations and changes in nuclear protein levels that accelerates the progression of alcoholic liver disease. A timely review is presented by Kirpich et al.

That provides the current understanding of recent advances regarding the role of dietary lipids in alcoholic liver disease pathogenesis. The review by Groebner and Tuma hypothesize that α-tubulin is a major target for modification by highly reactive ethanol metabolites and reactive oxygen species. They discuss the potential cellular consequences of microtubule modification in promoting hepatic dysfunction, with a focus on the alcohol-induced defects in protein trafficking and enhanced steatosis.

Two articles are devoted to autophagy and mitophagy as important protective mechanisms in alcoholic liver disease. One, an original article by Lu and Cederbaum presents elegant data obtained employing an ethanol fed mouse model system. Their study demonstrates that autophagy is protective against CYP2E1-dependent liver injury by minimizing the ethanol-induced CYP2E1-dependent oxidative stress and the development of steatosis and liver injury.

This indicates that attempts to stimulate autophagy may be helpful in lowering ethanol and CYP2E1-dependent liver toxicity. The second, an article by Williams and Ding presents an overview on the protective role of mitophagy in preventing alcohol-induced liver damage. Specifically, they discuss the roles of both Parkin-dependent and -independent mechanisms of mitophagy activation in protection against alcohol-induced liver injury and steatosis.

Udoh et al.’s review article on the role of impairment in the circadian clock in alcoholic liver disease development proposes that the alcohol-mediated disruption in circadian rhythms likely underpins many adverse health effects of alcohol that cut across multiple organ systems.

The authors summarize various molecular events by which alcohol may negatively impact circadian clock-mediated processes in the liver, thereby contributing to tissue pathology. The article by Szary et al. devoted to high intrinsic endurance/aerobic capacity presents a connection between high aerobic fitness and protection from metabolic diseases.

This study, using a high capacity runner rat model, demonstrates that ethanol ingestion fails to induce significant hepatic liver injury when different parameters such as hepatic inflammation or serum alanine amino transferase, free fatty acids, triglycerides, insulin and glucose levels were measured.

• While high intrinsic aerobic fitness protected against ethanol-induced hepatic injury and systemic metabolic dysfunction, it did not reduce ethanol-activated hepatic steatosis.
• The review by Neuman et al.
• Elucidates the mechanisms by which alcohol contributes to the activation of Kupffer cells and the inflammatory cascade and deliberates the role of the stellate cells in fibrogenesis.

Another article by Nanau and Neuman addresses the diagnostic value of biomolecules and biomarkers in alcohol drinking, especially in monitoring therapeutic interventions. The study by Harrison-Findik and Lu investigates the regulation of the key iron-regulatory molecule, hepcidin, by alcohol and hydrogen peroxide (H 2 O 2 ).

Employing glutathione peroxidase-1 (gpx-1 −/− ) and catalase (catalase −/− ) knockout mice, the authors conclude that H 2 O 2 inhibits hepcidin expression in vivo and that synergistic induction of CCAAT-enhancer-binding protein homologous protein (CHOP) by alcohol and H 2 O 2, in the absence of gpx-1, stimulates liver hepcidin gene expression by endoplasmic reticulum stress independent of CREBH.

The review “Update on Alcoholic Hepatitis” by Torok presents recent advances in the diagnosis, pathogenesis of alcoholic hepatitis and novel treatment strategies. The study by Scheer et al. demonstrates that acetaldehyde has a major role in the ethanol metabolism-mediated G2/M cell cycle arrest, and the concurrent accumulation of p21 (a cyclin-dependent kinase inhibitor) and p-Cdc2 (cell divison cycle protein 2 homolog).

• Although reactive oxygen species are thought to have a significant role in ethanol-induced hepatocellular damage, they may have a less important role in the inability of hepatocytes to replace dead or damaged cells.
• An article by Deshpande et al.
• Highlights previously unknown effects of moderate ethanol exposure on hepatic wound healing after acute carbon tetrachloride exposure by indicating that moderate ethanol affected each phase of the wound healing response to this hepatotoxicant.

Finally, the review article by Gitto et al. discusses the usefulness of psychosocial support in the management of people affected by alcohol. These authors reflect that a multidisciplinary approach involving clinical-psychologists, psychiatrics and hepatologists should be considered as essential in the management of patients with alcohol liver disease especially in the case of liver transplantation.

1. Several studies/reviews present an association between the ethanol-induced gut dysfunctions and the pathogenesis of liver injury,
2. Patel et al.
3. Review the mechanisms of alcohol-induced endotoxemia (including dysbiosis and gut leakiness) as well as the predisposing factors that promote these changes.
4. They also describe various barriers, including immunologic, physical, and biochemical that regulate the passage of toxins into the portal and systemic circulation.

In addition, the review by Massey et al. discusses the cross-talk between gut, liver and lung and the parallel mechanisms of liver and lung injury in response to alcohol consumption. The authors also explore the potential that these mechanisms are interdependent, as part of a gut-liver-lung axis The study by Zhong et al.

Illustrates that while alcohol consumption causes nicotinic acid deficiency, its supplementation upregulates the intestinal genes involved in aldehyde detoxification via transcriptional regulation. This indicates that the inhibitory effects of nicotinic acid on alcohol-induced endotoxemia and hepatic inflammation are via the modulation of the intestinal barrier function and bacterial endotoxin production.

Two articles reveal the mechanism of alcohol-induced lung damage. One article by Sapkota and Wyatt presents an overview on the formation of reactive aldehydes in the lung as a result of drinking alcohol and smoking cigarettes that react with nucleophilic targets in cells such as DNA, lipids and proteins to form both stable and unstable adducts.

The review provides an insight into different reactive aldehyde adducts and their role in the pathogenesis of lung disease. The second, a review by Traphagen et al. follows the influence of alcohol on lung injury and the role of ethanol exposure in the pathogenesis and progression of pulmonary disease.

Alcohol is also shown to affect heart function. The article “Alcohol and Apoptosis: Friends or Foes?” by Rodriguez et al. is devoted to the in vivo effects of alcohol exposure on cardiomyocytes contraction and relaxation. The authors demonstrate that the animals on high doses of alcohol display a marked thinning of the left ventricular wall along with elevated caspase-3 activity and decreased contractility.

In contrast, low alcohol is associated with increased contractility and decreased apoptosis suggesting that overall protective mechanisms are induced by low levels of alcohol exposure. Brand et al. contributed to this Special Issue with the hypotheses that the skin immune network may be relatively preserved in alcohol consumers and proposed that the development of skin-targeted immunizations could circumvent the immune inhibitory effects of alcohol consumption.

The authors demonstrate that independent of the feeding model, ethanol ingestion inhibits delayed type hypersensitivity, lysis by cytotoxic T-lymphocytes, and antigen-specific total IgG induced by traditional systemic vaccines. They further show that skin-targeted vaccines were equally immunogenic in alcohol-exposed and non-exposed subjects suggesting that cutaneous immunization may result in more efficacious vaccination in alcohol abusing subjects.

Only one study was related to neurotoxic effects of alcohol. To this end, Yang and Luo review the role of ethanol-induced endoplasmic reticulum stress on neurotoxicity. The authors discuss recent progress in the study of endoplasmic reticulum stress in ethanol-induced neurotoxicity and examine the interaction among endoplasmic reticulum stress, oxidative stress and autophagy in the context of ethanol neurotoxicity.

Overall, this Special Issue on “Multi-Organ Alcohol-Related Damage: Mechanisms and Treatment” covers important aspects on alcohol-induced organ damage and provides comprehensive information on the mechanisms underlying these events. The published articles are recommended for scientists and physicians involved in basic, translational and/or clinical studies on alcohol-induced injury to cells, tissues and multiple organs.

The list of articles published in Special Issue “Multi-Organ Alcohol-Related Damage: Mechanisms and Treatment” is presented in Refs. The authors acknowledge the use of facilities at the VA Nebraska-Western Iowa Health Care System and funding (Merit Review Grants BX001673 and BX001155 ) from the Department of Veterans Affairs, Office of Research and Development (Biomedical Laboratory Research and Development).

The authors declare no conflict of interest.1. Natarajan S., Pachunka J., Mott J. Role of microRNAs in Alcohol-Induced Multi-Organ Injury. Biomolecules.2015; 5 :3309–3338. doi: 10.3390/biom5043309. Available online:,2. Anji A., Kumari M. Guardian of Genetic Messenger-RNA-Binding Proteins.

1. Biomolecules.2016; 6 :4.
2. Doi: 10.3390/biom6010004.
3. Available online:,3.
4. Steiner J., Crowell K., Lang C.
5. Impact of Alcohol on Glycemic Control and Insulin Action.
6. Biomolecules.2015; 5 :2223–2246.
7. Doi: 10.3390/biom5042223.
8. Available online:,4. Ji C.
9. Advances and New Concepts in Alcohol-Induced Organelle Stress, Unfolded Protein Responses and Organ Damage.

Biomolecules.2015; 5 :1099–1121. doi: 10.3390/biom5021099. Available online:,5. Shukla S., Aroor A., Restrepo R., Kharbanda K., Ibdah J. In Vivo Acute on Chronic Ethanol Effects in Liver: A Mouse Model Exhibiting Exacerbated Injury, Altered Metabolic and Epigenetic Responses.

Biomolecules.2015; 5 :3280–3294. doi: 10.3390/biom5043280. Available online:,6. Kirpich I., Miller M., Cave M., Joshi-Barve S., McClain C. Alcoholic Liver Disease: Update on the Role of Dietary Fat. Biomolecules.2016; 6 :1. doi: 10.3390/biom6010001. Available online:,7. Groebner J., Tuma P. The Altered Hepatic Tubulin Code in Alcoholic Liver Disease.

Biomolecules.2015; 5 :2140–2159. doi: 10.3390/biom5032140. Available online:,8. Lu Y., Cederbaum A. Autophagy Protects against CYP2E1/Chronic Ethanol-Induced Hepatotoxicity. Biomolecules.2015; 5 :2659–2674. doi: 10.3390/biom5042659. Available online:,9. Williams J., Ding W.

A Mechanistic Review of Mitophagy and Its Role in Protection against Alcoholic Liver Disease. Biomolecules.2015; 5 :2619–2642. doi: 10.3390/biom5042619. Available online:,10. Udoh U., Valcin J., Gamble K., Bailey S. The Molecular Circadian Clock and Alcohol-Induced Liver Injury. Biomolecules.2015; 5 :2504–2537.

doi: 10.3390/biom5042504. Available online:,11. Szary N., Rector R., Uptergrove G., Ridenhour S., Shukla S., Thyfault J., Koch L., Britton S., Ibdah J. High Intrinsic Aerobic Capacity Protects against Ethanol-Induced Hepatic Injury and Metabolic Dysfunction: Study Using High Capacity Runner Rat Model.

1. Biomolecules.2015; 5 :3295–3308.
2. Doi: 10.3390/biom5043295.
3. Available online:,12.
4. Neuman M., Maor Y., Nanau R., Melzer E., Mell H., Opris M., Cohen L., Malnick S.
5. Alcoholic Liver Disease: Role of Cytokines.
6. Biomolecules.2015; 5 :2023–2034.
7. Doi: 10.3390/biom5032023.
8. Available online:,13.
9. Nanau R., Neuman M.

Biomolecules and Biomarkers Used in Diagnosis of Alcohol Drinking and in Monitoring Therapeutic Interventions. Biomolecules.2015; 5 :1339–1385. doi: 10.3390/biom5031339. Available online:,14. Harrison-Findik D., Lu S. The Effect of Alcohol and Hydrogen Peroxide on Liver Hepcidin Gene Expression in Mice Lacking Antioxidant Enzymes, Glutathione Peroxidase-1 or Catalase.

Biomolecules.2015; 5 :793–807. doi: 10.3390/biom5020793. Available online:,15. Torok N. Update on Alcoholic Hepatitis. Biomolecules.2015; 5 :2978–2986. doi: 10.3390/biom5042978. Available online:,16. Scheer M.A., Schneider K.J., Finnigan R.L., Maloney E.P., Wells M.A., Clemens D.L. The Involvement of Acetaldehyde in Ethanol-Induced Cell Cycle Impairment.

Biomolecules.2016; 6 :17. doi: 10.3390/biom6020017. Available online:,17. Deshpande K., Liu S., McCracken J., Jiang L., Gaw T., Kaydo L., Richard Z., O’Neil M., Pritchard M. Moderate (2%, v/v ) Ethanol Feeding Alters Hepatic Wound Healing after Acute Carbon Tetrachloride Exposure in Mice.

1. Biomolecules.2016; 6 :5.
2. Doi: 10.3390/biom6010005.
3. Available online:,18.
4. Gitto S., Golfieri L., Caputo F., Grandi S., Andreone P.
5. Multidisciplinary View of Alcohol Use Disorder: From a Psychiatric Illness to a Major Liver Disease.
6. Biomolecules.2016; 6 :11.
7. Doi: 10.3390/biom6010011.
8. Available online:,19.
9. Patel S., Behara R., Swanson G., Forsyth C., Voigt R., Keshavarzian A.

Alcohol and the Intestine. Biomolecules.2015; 5 :2573–2588. doi: 10.3390/biom5042573. Available online:,20. Massey V., Beier J., Ritzenthaler J., Roman J., Arteel G. Potential Role of the Gut/Liver/Lung Axis in Alcohol-Induced Tissue Pathology. Biomolecules.2015; 5 :2477–2503.

• Doi: 10.3390/biom5042477.
• Available online:,21.
• Zhong W., Li Q., Zhang W., Sun Q., Sun X., Zhou Z.
• Modulation of Intestinal Barrier and Bacterial Endotoxin Production Contributes to the Beneficial Effect of Nicotinic Acid on Alcohol-Induced Endotoxemia and Hepatic Inflammation in Rats.
• Biomolecules.2015; 5 :2643–2658.

doi: 10.3390/biom5042643. Available online:,22. Sapkota M., Wyatt T. Alcohol, Aldehydes, Adducts and Airways. Biomolecules.2015; 5 :2987–3008. doi: 10.3390/biom5042987. Available online:,23. Traphagen N., Tian Z., Allen-Gipson D. Chronic Ethanol Exposure: Pathogenesis of Pulmonary Disease and Dysfunction.

Biomolecules.2015; 5 :2840–2853. doi: 10.3390/biom5042840. Available online:,24. Rodriguez A., Chawla K., Umoh N., Cousins V., Ketegou A., Reddy M., AlRubaiee M., Haddad G., Burke M. Alcohol and Apoptosis: Friends or Foes? Biomolecules.2015; 5 :3193–3203. doi: 10.3390/biom5043193. Available online:,25. Brand R., Stottlemyer J., Cline R., Donahue C., Behari J., Falo L., Jr.

Skin Immunization Obviates Alcohol-Related Immune Dysfunction. Biomolecules.2015; 5 :3009–3028. doi: 10.3390/biom5043009. Available online:,26. Yang F., Luo J. Endoplasmic Reticulum Stress and Ethanol Neurotoxicity. Biomolecules.2015; 5 :2538–2553. doi: 10.3390/biom5042538.

Should I stop drinking alcohol if my kidneys hurt?

– Moderate alcohol consumption should not cause kidney pain. However, excess alcohol consumption may injure the kidneys or increase the risk of chronic kidney disease, Kidney pain after drinking alcohol may be a sign of these conditions.

Why does my left kidney hurt a bit?

How is kidney pain treated? – The treatment for kidney pain depends on what is causing it. Be sure to call your doctor if you have any kidney pain. Your doctor may do:

A urine test to check for signs of infectionImaging tests, such as an ultrasound or CT scan, to see if your kidneys are injured

Once you know what is causing your pain, your doctor can work with you to find the right treatment. Webinar

Does alcohol damage kidneys or liver?

Abstract – Both acute and chronic alcohol consumption can compromise kidney function, particularly in conjunction with established liver disease. Investigators have observed alcohol-related changes in the structure and function of the kidneys and impairment in their ability to regulate the volume and composition of fluid and electrolytes in the body.

• Chronic alcoholic patients may experience low blood concentrations of key electrolytes as well as potentially severe alterations in the body’s acid-base balance.
• In addition, alcohol can disrupt the hormonal control mechanisms that govern kidney function.
• By promoting liver disease, chronic drinking has further detrimental effects on the kidneys, including impaired sodium and fluid handling and even acute kidney failure.

Keywords: kidney function, kidney disorder, disorder of fluid or electrolyte or acid-base balance, alcoholic liver disorder, hormones, body fluid, blood circulation, blood pressure, sodium, potassium, phosphates, magnesium, calcium, literature review A cell’s function depends not only on receiving a continuous supply of nutrients and eliminating metabolic waste products but also on the existence of stable physical and chemical conditions in the extracellular fluid 1 bathing it.

Among the most important substances contributing to these conditions are water, sodium, potassium, calcium, and phosphate. Loss or retention of any one of these substances can influence the body’s handling of the others. In addition, hydrogen ion concentration (i.e., acid-base balance) influences cell structure and permeability as well as the rate of metabolic reactions.

The amounts of these substances must be held within very narrow limits, regardless of the large variations possible in their intake or loss. The kidneys are the organs primarily responsible for regulating the amounts and concentrations of these substances in the extracellular fluid.

1. In addition to their role in regulating the body’s fluid composition, the kidneys produce hormones that influence a host of physiological processes, including blood pressure regulation, red blood cell production, and calcium metabolism.
2. Besides producing hormones, the kidneys respond to the actions of regulatory hormones produced in the brain, the parathyroid glands in the neck, and the adrenal glands located atop the kidneys.

Because of the kidneys’ important and varied role in the body, impairment of their function can result in a range of disorders, from mild variations in fluid balance to acute kidney failure and death. Alcohol, one of the numerous factors that can compromise kidney function, can interfere with kidney function directly, through acute or chronic consumption, or indirectly, as a consequence of liver disease.

When should I worry about left side pain?

When should I worry about left side pain? – Generally, intermittent left side pain is a sign of gas or indigestion and should pass on its own. But if you notice severe abdominal pain on the left side of your body in conjunction with any of the following symptoms, it’s time to seek medical help:

FeverNausea or vomitingCold or clammy skinRapid breathingDizzy or lightheadednessMuscle weaknessBloody stools or vomitAbdominal pain that lasts more than a week

When I drink alcohol my lower left abdomen hurts?

What is alcoholic gastritis? – Gastritis means inflammation of the stomach. The word gastritis comes from gastric which means “of the stomach” and itis which means “inflammation”. The walls of our stomachs are sensitive. If we expose them to excessive volumes of alcohol, they can become irritated and damaged.

Why does the left side of my left rib hurt?

What kind of doctor should I see for left rib pain? – There are many possible causes of pain around your left rib cage. A primary care doctor can usually help you determine the cause. If needed, they can refer you to a specialist, such as an orthopedic doctor who treats bones, muscles, and joints.

What are the 3 early warning signs of liver disease?

Symptoms – Liver disease doesn’t always cause noticeable signs and symptoms. If signs and symptoms of liver disease do occur, they may include:

Skin and eyes that appear yellowish (jaundice) Abdominal pain and swelling Swelling in the legs and ankles Itchy skin Dark urine color Pale stool color Chronic fatigue Nausea or vomiting Loss of appetite Tendency to bruise easily

Which organ does alcohol hurt the most?

Drinking too much – on a single occasion or over time – can take a serious toll on your health. Here’s how alcohol can affect your body: Brain: Alcohol interferes with the brain’s communication pathways, and can affect the way the brain looks and works.

Cardiomyopathy – Stretching and drooping of heart muscle Arrhythmias – Irregular heart beat Stroke High blood pressure

Liver: Heavy drinking takes a toll on the liver, and can lead to a variety of problems and liver inflammations including:

Steatosis, or fatty liver Alcoholic hepatitis Fibrosis Cirrhosis

Pancreas: Alcohol causes the pancreas to produce toxic substances that can eventually lead to pancreatitis, a dangerous inflammation and swelling of the blood vessels in the pancreas that prevents proper digestion. Cancer: According to the National Cancer Institute: “There is a strong scientific consensus that alcohol drinking can cause several types of cancer.

In its Report on Carcinogens, the National Toxicology Program of the US Department of Health and Human Services lists consumption of alcoholic beverages as a known human carcinogen. “The evidence indicates that the more alcohol a person drinks–particularly the more alcohol a person drinks regularly over time–the higher his or her risk of developing an alcohol-associated cancer.

Even those who have no more than one drink per day and people who binge drink (those who consume 4 or more drinks for women and 5 or more drinks for men in one sitting) have a modestly increased risk of some cancers. Based on data from 2009, an estimated 3.5% of cancer deaths in the United States (about 19,500 deaths were alcohol related.” Clear patterns have emerged between alcohol consumption and increased risks of certain types of cancer:

Head and neck cancer, including oral cavity, pharynx, and larynx cancers.

Esophageal cancer, particularly esophageal squamous cell carcinoma. In addition, people who inherit a deficiency in an enzyme that metabolizes alcohol have been found to have substantially increased risks of esophageal squamous cell carcinoma if they consume alcohol.

Liver cancer.

Breast cancer: Studies have consistently found an increased risk of breast cancer in women with increasing alcohol intake. Women who consume about 1 drink per day have a 5 to 9 percent higher chance of developing breast cancer than women who do not drink at all.

Colorectal cancer.

For more information about alcohol and cancer, please visit the National Cancer Institute’s webpage ” Alcohol and Cancer Risk ” (last accessed October 21, 2021). Immune System: Drinking too much can weaken your immune system, making your body a much easier target for disease.

Will liver pain go away?

– Many conditions can cause liver pain, so it is important to find out which one is present. A doctor will carry out a physical examination and look at the person’s health history. Other testing options may include:

blood tests to assess liver function or identify specific liver problems or genetic conditionsimaging tests, such as CT scans, MRIs, and ultrasounds to assess for liver damagetissue analysis, which involves taking a tissue sample from the liver for a lab test

Sometimes, liver pain will go away after making some lifestyle changes, such as abstaining from alcohol, losing weight, or following a healthful diet. Other problems may require medication or surgery. If there is liver failure, a liver transplant may be needed. What are some other causes of abdominal pain? Find out here.

What organ hurts after heavy drinking?

Types of Alcohol Related Liver Disease –

Alcholol Related Steatohepatitis (ASH): Fat accumulates inside liver cells, making it hard for the liver to work properly. This early stage of liver disease occurs fairly soon after repeated heavy drinking. Usually it is symptom free but upper abdominal pain on the right side from an enlarged liver may occur. Steatosis goes away with alcohol abstinence. Alcoholic Hepatitis: This condition is marked by inflammation, swelling and the killing of liver cells. This scars the liver, which is known as fibrosis. Symptoms may occur over time or suddenly after binge drinking. They include fever, jaundice, nausea, vomiting, abdominal pain and tenderness. Up to 35 percent of heavy drinkers develop alcohol hepatitis, which can be mild or severe. If it is a mild case, stopping the drinking can reverse it. Alcohol Related Cirrhosis : The most serious form of ALD, it occurs when the entire liver is scarred, causing the liver to shrink and harden. This can lead to liver failure. Usually the damage cannot be reversed. Between 10 to 20 percent of heavy drinkers develop cirrhosis typically after 10 or more years of drinking.

Alcohol hepatitis and alcohol cirrhosis previously were called alcohol steatohepatitis (ASH), a term that still arises among some circles.

How long does liver pain last after drinking?

What is alcoholic hepatitis? – Alcoholic hepatitis is an inflammation of the liver that lasts one to two weeks.It is believed to lead to alcoholic cirrhosis over a period of years. Symptoms include of alcoholic hepatitis include:

Loss of appetite Nausea Vomiting Abdominal pain and tenderness Fever Jaundice Mental confusion

When I drink alcohol my right side back hurts?

Effect of Drinking Alcohol on Your Kidneys – Your kidneys play a very important role in keeping your body healthy and free from harmful substances. Aside from filtering and getting rid of the waste, your kidneys also make sure that your body maintains a proper balance of fluid and electrolytes.

So when you’re drinking, your kidneys work double-time to flush out the excess alcohol from your body, which can result in the pain that you’re feeling right now. When you drink alcohol, you tend to urinate frequently, which leads to dehydration. This can affect your kidney’s functions, as well as the other organs.

This gives way to symptoms such as pain in your kidney, flank, and back.

What organ hurts when you drink too much alcohol?

Drinking too much – on a single occasion or over time – can take a serious toll on your health. Here’s how alcohol can affect your body: Brain: Alcohol interferes with the brain’s communication pathways, and can affect the way the brain looks and works.

Cardiomyopathy – Stretching and drooping of heart muscle Arrhythmias – Irregular heart beat Stroke High blood pressure

Liver: Heavy drinking takes a toll on the liver, and can lead to a variety of problems and liver inflammations including:

Steatosis, or fatty liver Alcoholic hepatitis Fibrosis Cirrhosis

Pancreas: Alcohol causes the pancreas to produce toxic substances that can eventually lead to pancreatitis, a dangerous inflammation and swelling of the blood vessels in the pancreas that prevents proper digestion. Cancer: According to the National Cancer Institute: “There is a strong scientific consensus that alcohol drinking can cause several types of cancer.

• In its Report on Carcinogens, the National Toxicology Program of the US Department of Health and Human Services lists consumption of alcoholic beverages as a known human carcinogen.
• The evidence indicates that the more alcohol a person drinks–particularly the more alcohol a person drinks regularly over time–the higher his or her risk of developing an alcohol-associated cancer.

Even those who have no more than one drink per day and people who binge drink (those who consume 4 or more drinks for women and 5 or more drinks for men in one sitting) have a modestly increased risk of some cancers. Based on data from 2009, an estimated 3.5% of cancer deaths in the United States (about 19,500 deaths were alcohol related.” Clear patterns have emerged between alcohol consumption and increased risks of certain types of cancer:

Head and neck cancer, including oral cavity, pharynx, and larynx cancers.

Esophageal cancer, particularly esophageal squamous cell carcinoma. In addition, people who inherit a deficiency in an enzyme that metabolizes alcohol have been found to have substantially increased risks of esophageal squamous cell carcinoma if they consume alcohol.

Liver cancer.

Breast cancer: Studies have consistently found an increased risk of breast cancer in women with increasing alcohol intake. Women who consume about 1 drink per day have a 5 to 9 percent higher chance of developing breast cancer than women who do not drink at all.

Colorectal cancer.

For more information about alcohol and cancer, please visit the National Cancer Institute’s webpage ” Alcohol and Cancer Risk ” (last accessed October 21, 2021). Immune System: Drinking too much can weaken your immune system, making your body a much easier target for disease.

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