How is Methanol Produced in Moonshine? – via GIPHY Methanol is a by-product produced during the fermentation process of making moonshine. During fermentation the enzymes in the yeast are responsible for converting the fermentable sugars into energy for the cell.
Contents
- 1 How do you prevent methanol in homebrew?
- 2 How do distillers remove methanol?
- 3 Is methanol OK to drink?
- 4 How much methanol is toxic?
- 5 Can you cure methanol poisoning with alcohol?
How do you prevent methanol when making moonshine?
If you love your moonshine, you might be curious about the distillation process and want to try it yourself. You’ve probably also heard horror stories about people who get sick and even die from methanol poisoning. To distill moonshine without any methanol, you must discard the first portion of your distillate containing poisonous compounds.
Why do you get methanol when distilling?
During fruit sugar fermentation to ethanol by yeast, methanol is formed primarily through degradation of pectin by pectin-methylesterase (PME).
Can moonshine cause methanol poisoning?
Moonshine is related to southern culture, heritage, and tradition, but its origin can be traced back to Pennsylvania. Around 1791, when the distilled-spirits tax was implemented, farmers grew frustrated and were determined to do something about the matter.
How do you prevent methanol in homebrew?
2.1.3. Inhibition of Pectin Methylesterase by Sterilization of Mash – A significant reduction of methanol by 40–90% can be achieved by thermal deactivation of pectin methylesterase (often referred to as “mash heating”). There are various suggestions for temperature/time combinations to achieve the enzyme’s denaturation.
Sterilization at temperatures higher than 70 °C was generally suggested to effectively prevent the production of methanol by inactivation of pectin methylesterase, Methanol can be reduced by targeted thermal deactivation of pectin methylesterase by heating the mash to 80 °C up to 85 °C for a holding time of 30 min or to 60 °C for 45 min,
Pasteurization at 72 °C for 15 s prevented the production of methanol in fermented plant beverages containing Morinda citrifolia (noni fruit), In cider spirit, the pasteurization (30 min at 50 °C, then heated to about 85 °C) of the apple juice prior to fermentation reduced the methanol content by 34–46%,
Lower methanol levels were obtained in Williams and plums by heating the mash to 65 °C for 5 min, followed by re-cooling for fermentation, Xia et al. confirmed that autoclaving by steam injection of the mash of jujube reduced the methanol content in the spirit significantly by a factor of about eight.
The authors also determined pectin methylesterase activity confirming that their treatment method reduced the activity to one-fifth to half of that without treatment. Further technological approaches for inactivation of methylesterase are thermosonication (ultrasound plus temperature at 70° led to 30% methanol reduction in plum wine) or use of microwaves (70 °C for 1 min led to 70% methanol reduction in plum wine).
How do distillers remove methanol?
How to separate methanol in distillation – Methanol is more volatile than ethanol under all operating conditions adopted by distilleries, so it is possible to separate it by distillation as “heads” or “head product”. In the case of a batch distillation plant, we separate methanol using an appropriate choice of head-cut timing.
- The longer the timing of the cut, the more methanol is removed, but in the process some of the “heart” is lost.
- It is therefore essential to find the balance between maximum methanol removal and maximum amount of heart produced.
- In a continuous column or plant it is possible to take a stream of “heads” rich in methanol, taking it from the reflux of the column.
Since in these plants the distillate is usually at higher alcohol concentrations than in a batch plant, and the methanol purity specifications are often more stringent, a demethylating column is often used. While in the demethylating column we have the distillate stream at high alcohol concentration.
- Inside it, the separation between methanol (head product) and ethanol (tail product) takes place.
- Because of the similar volatility rate of methanol and ethanol, the tail product (demethylated distillate) will contain traces of methanol in concentrations below the required specifications, whereas the head product, rich in methanol, will contain significant quantities of ethanol.
The latter percentage of ethanol is a by-product of distillation. It should be recovered to avoid waste and improve the overall efficiency of the distillery.
How do you know if alcohol has methanol in it?
Introduction – Toxic alcohol consumption is a major cause of mortalities and morbidities worldwide, Although drinking alcohol is prohibited in Muslim countries and there have been major penalties determined for alcohol use in them, recent statistics show that these penalties have failed to decrease the frequency of alcohol use or misuse in some of them,
- This has resulted in increased use of black market alcohol which may potentially be methanol-contaminated due to the lack of observatory quality control processes and outbreaks of methanol poisoning in different parts of the world,
- Considering the Eastern Mediterranean Region (EMR) as the region with Islamic countries within, both men and women in this area have the highest weekly heavy episodic drinking among drinkers in the past 12 months in both males and females worldwide,
The worldwide consumption of ethanol was equal to 6.13 l of pure alcohol consumed per person of 15 years of age or older in 2005. A large portion of this consumption – 28.6% or 1.76 l per person – was homemade, illegally produced or sold outside normal government controls,
- This increases the risk of introduction of hazardous chemicals into the ethanol, the most important of which is methanol,
- Both unsupervised production of alcoholic beverages and lack of quality control processes during their production increase the risk of contamination of the produced alcohol with unwanted toxic components including methanol.
Therefore, during the process of quality control of production of such beverages, it is generally important to be able to determine the presence of sufficient methanol concentration capable of resulting in poisoning. Police were usually asked to investigate the discovered consignment of suspected alcoholic beverages and report its content to the judiciary system to determine their alcohol concentration.
Based on Iranian legal medicine organization protocols, liquids with 3% v/v ethanol or less than that are not legally considered to be alcoholic beverage at all. The gold standard method for determination of methanol content in alcoholic beverages is gas chromatography (GC). However, this technique is expensive, calls for considerable knowledge and experience to be performed, and is not readily available in many developing countries although this technique has previously been used even in mass poisonings,
Having access to a safe, cheap and easy method to prove the absence of unauthorized quantities of methanol before ingestion is therefore highly advantageous, Generally, with the same methanol concentration, the possibility of toxicity increases with reduced ethanol content.
Ethanol has a 20 times higher affinity for liver alcohol dehydrogenase enzyme which prevents methanol metabolism when blood ethanol level is 100 mg/dL or higher, Previous studies declare up to 5 mg/dL serum methanol level as the acceptable concentration of this toxic agent in human blood, Reaching this methanol level in an average 75-kg adult with about 41 l of body water (55% of the total body weight) would roughly be possible after consuming 251 mg methanol in 1–2 h.
This is approximately equal to 2.5% v/v absolute methanol in water, Thus, determination of the maximum acceptable methanol to ethanol concentration in an alcoholic drink without risking toxicity is a challenging concern. The “maximum safe” concentration of methanol in alcoholic beverages has previously been determined based on “permitted and safe content of methanol in the beverages” regulated by the European Parliament and the Council (4000 mg/L in alcoholic drinks with 40% v/v ethanol concentration) and US national research council of the national academies (Table 1 ),
Therefore, “maximum safe dose” is defined to avoid a serum methanol concentration more than 5 mg/dL, Table 1 Methanol Concentrations in Food and Beverages We used a new kit designed based on the modified chromotropic acid (CA) method for this purpose. Using this kit, the relative concentration of methanol to ethanol is estimated since methanol/ethanol ratio can predict the potency of the drink to induce methanol toxicity.
Therefore, a positive test would indicate an unsafe beverage and the possibility of methanol poisoning. We picked a conservative approach to evaluate the potency for both acute and chronic methanol toxicities. The table for safe concentration of methanol in different food products and beverages (USA standard) was therefore used (Table 1 ) which determined all drinks with any concentration below the permitted levels as safe beverages.
Preliminary evaluations confirmed the efficacy of this kit in determination of possible toxicity risk of the alcoholic beverages, The aim of the current study was to firstly evaluate the methanol and ethanol contents of the suspected alcoholic beverages discovered by Iranian police as sample of the alcoholic beverages available in the Iranian black market using GC as the gold standard method.
As a second aim, we assessed the potency of toxicity of these suspected samples by detection of relative methanol to ethanol content using a new kit based on modified CA method and compared them with the results obtained by GC in order to determine the efficacy of the designed kit.
Why is the first cup of moonshine toxic?
Methanol: The Toxic Side Of Moonshine – You may have heard stories about the dangers of moonshine, or any illegally distilled liquor. The real culprit of these true stories, however, is methanol. Methanol, or methyl alcohol, is a byproduct of the distilling process.
Does fermenting sugar produce methanol?
When fermenting any kind of sugar water solution then there are small quantities of by-products such as methanol produced. For the most part, this is no issue, however when you distil and concentrate the alcohols in the wash (including methanol) then it can become an issue.
Is methanol OK to drink?
Methanol: Systemic Agent
CAS #: 67-56-1 RTECS #: PC1400000 UN #: 1230 (Guide 131)
Common Names:
Carbinol Methyl alcohol Wood alcohol
APPEARANCE : Colorless watery liquid. DESCRIPTION : Methanol is a toxic alcohol that is used industrially as a solvent, pesticide, and alternative fuel source. It also occurs naturally in humans, animals, and plants. Foods such as fresh fruits and vegetables, fruit juices, fermented beverages, and diet soft drinks containing aspartame are the primary sources of methanol in the human body. Most methanol poisonings occur as a result of drinking beverages contaminated with methanol or from drinking methanol-containing products. In the industrial setting, inhalation of high concentrations of methanol vapor and absorption of methanol through the skin are as effective as the oral route in producing toxic effects. The characteristic pungent (alcohol) odor of methanol does not provide sufficient warning of low levels of exposure. METHODS OF DISSEMINATION :
Indoor Air: Methanol can be released into indoor air as a liquid spray (aerosol). Water: Methanol can be used to contaminate water. Food: Methanol may be used to contaminate food. Outdoor Air: Methanol can be released into outdoor air as a liquid spray (aerosol). Agricultural: If methanol is released into the air as a liquid spray (aerosol), it has the potential to contaminate agricultural products.
ROUTES OF EXPOSURE : Methanol can be absorbed into the body by inhalation, ingestion, skin contact, or eye contact. Ingestion is an important route of exposure.
GENERAL INFORMATION : First Responders should use a NIOSH-certified Chemical, Biological, Radiological, Nuclear (CBRN) Self Contained Breathing Apparatus (SCBA) with a Level A protective suit when entering an area with an unknown contaminant or when entering an area where the concentration of the contaminant is unknown. Level A protection should be used until monitoring results confirm the contaminant and the concentration of the contaminant. NOTE: Safe use of protective clothing and equipment requires specific skills developed through training and experience. LEVEL A: (RED ZONE) : Select when the greatest level of skin, respiratory, and eye protection is required. This is the maximum protection for workers in danger of exposure to unknown chemical hazards or levels above the IDLH or greater than the AEGL-2.
A NIOSH-certified CBRN full-face-piece SCBA operated in a pressure-demand mode or a pressure-demand supplied air hose respirator with an auxiliary escape bottle. A Totally-Encapsulating Chemical Protective (TECP) suit that provides protection against CBRN agents. Chemical-resistant gloves (outer). Chemical-resistant gloves (inner). Chemical-resistant boots with a steel toe and shank. Coveralls, long underwear, and a hard hat worn under the TECP suit are optional items.
LEVEL B: (RED ZONE) : Select when the highest level of respiratory protection is necessary but a lesser level of skin protection is required. This is the minimum protection for workers in danger of exposure to unknown chemical hazards or levels above the IDLH or greater than AEGL-2.
A NIOSH-certified CBRN full-face-piece SCBA operated in a pressure-demand mode or a pressure-demand supplied air hose respirator with an auxiliary escape bottle. A hooded chemical-resistant suit that provides protection against CBRN agents. Chemical-resistant gloves (outer). Chemical-resistant gloves (inner). Chemical-resistant boots with a steel toe and shank. Coveralls, long underwear, a hard hat worn under the chemical-resistant suit, and chemical-resistant disposable boot-covers worn over the chemical-resistant suit are optional items.
LEVEL C: (YELLOW ZONE) : Select when the contaminant and concentration of the contaminant are known and the respiratory protection criteria factors for using Air Purifying Respirators (APR) or Powered Air Purifying Respirators (PAPR) are met. This level is appropriate when decontaminating patient/victims.
A NIOSH-certified CBRN tight-fitting APR with a canister-type gas mask or CBRN PAPR for air levels greater than AEGL-2. A NIOSH-certified CBRN PAPR with a loose-fitting face-piece, hood, or helmet and a filter or a combination organic vapor, acid gas, and particulate cartridge/filter combination or a continuous flow respirator for air levels greater than AEGL-1. A hooded chemical-resistant suit that provides protection against CBRN agents. Chemical-resistant gloves (outer). Chemical-resistant gloves (inner). Chemical-resistant boots with a steel toe and shank. Escape mask, face shield, coveralls, long underwear, a hard hat worn under the chemical-resistant suit, and chemical-resistant disposable boot-covers worn over the chemical-resistant suit are optional items.
LEVEL D: (GREEN ZONE) : Select when the contaminant and concentration of the contaminant are known and the concentration is below the appropriate occupational exposure limit or less than AEGL-1 for the stated duration times.
Limited to coveralls or other work clothes, boots, and gloves.
CHEMICAL DANGERS :
Methanol reacts violently with strong oxidants, causing a fire and explosion hazard.
EXPLOSION HAZARDS :
Mixtures of methanol vapor and air are explosive. Lower explosive (flammable) limit in air (LEL), 6.0%; upper explosive (flammable) limit in air (UEL), 36%. Agent presents a vapor explosion and poison (toxic) hazard indoors, outdoors, or in sewers. Run-off to sewers may create an explosion hazard. Containers may explode when heated.
FIRE FIGHTING INFORMATION :
Methanol is highly flammable. The agent will be easily ignited by heat, sparks, or flames. Fire will produce irritating, corrosive, and/or toxic gases. Vapors may travel to the source of ignition and flash back. Run-off to sewers may create a fire hazard. Caution: The agent has a very low flash point. Use of water spray when fighting fires may be inefficient. For small fires, use dry chemical, carbon dioxide, water spray, or alcohol-resistant foam. For large fires, use water spray, fog, or alcohol-resistant foam. Move containers from the fire area if it is possible to do so without risk to personnel. Dike fire control water for later disposal; do not scatter the agent. Use water spray or fog; do not use straight streams. For fire involving tanks or car/trailer loads, fight the fire from maximum distance or use unmanned hose holders or monitor nozzles. Cool containers with flooding quantities of water until well after the fire is out. Withdraw immediately in case of rising sound from venting safety devices or discoloration of tanks. Always stay away from tanks engulfed in fire. For massive fire, use unmanned hose holders or monitor nozzles; if this is impossible, withdraw from the area and let the fire burn. Run-off from fire control or dilution water may cause pollution. If the situation allows, control and properly dispose of run-off (effluent).
INITIAL ISOLATION AND PROTECTIVE ACTION DISTANCES :
If a tank, rail car, or tank truck is involved in a fire, isolate it for 0.5 mi (800 m) in all directions; also consider initial evacuation for 0.5 mi (800 m) in all directions. This agent is not included in the DOT ERG 2004 Table of Initial Isolation and Protective Action Distances. In the DOT ERG 2004 orange-bordered section of the guidebook, there are public safety recommendations to isolate a methanol (Guide 131) spill or leak area immediately for at least 150 ft (50 m) in all directions.
PHYSICAL DANGERS :
Methanol vapors may be heavier than air. They will spread along the ground and collect and stay in poorly-ventilated, low-lying, or confined areas (e.g., sewers, basements, and tanks). Hazardous concentrations may develop quickly in enclosed, poorly-ventilated, or low-lying areas. Keep out of these areas. Stay upwind. Liquid agent is lighter than water.
NFPA 704 Signal :
Health: 1 Flammability: 3 Reactivity: 0 Special:
SAMPLING AND ANALYSIS :
OSHA: 91 NIOSH: 2000, 3800
ADDITIONAL SAMPLING AND ANALYSIS INFORMATION : References are provided for the convenience of the reader and do not imply endorsement by NIOSH.
AIR MATRIX Allen TM, Falconer TM, Cisper ME, Borgerding AJ, Wilkerson CW Jr., Real-time analysis of methanol in air and water by membrane introduction mass spectrometry. Anal Chem 73(20):4830-4835.De Paula PP, Santos E, De Freitas FT, De Andrade JB, Determination of methanol and ethanol by gas chromatography following air sampling onto florisil cartridges and their concentrations at urban sites in the three largest cities in Brazil. Talanta 49(2):245-252. Leibrock E, Slemr J, Method for measurement of volatile oxygenated hydrocarbons in ambient air. Atmos Environ 31(20):3329-3339. Marley NA, Gaffney JS, A comparison of flame ionization and ozone chemiluminescence for the determination of atmospheric hydrocarbons. Atmos Environ 32(8):1435-1444. NIOSH, NMAM Method 2000 Methanol. In: NIOSH Manual of analytical methods.4th ed. Cincinnati, OH: U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, DHHS (NIOSH) Publication 94-113. OSHA, Methyl Alcohol Method 91. Salt Lake City, UT.U.S. Department of Labor, Organic Methods Evaluation Branch, OSHA Salt Lake Technical Center. Qin T, Xu X, Polak T, Pacakova V, Stulik K, Jech L, A simple method for the trace determination of methanol, ethanol, acetone, and pentane in human breath and in the ambient air by preconcentration on solid sorbents followed by gas chromatography. Talanta 44(9):1683-1690. Reichert J, Coerdt W, Ache HJ, Development of a surface acoustic wave sensor array for the detection of methanol in fuel vapours. Sens Actuators B: Chem 13(1-3):293-296. Tyras H, Spectrophotometric determination of methyl alcohol in the atmosphere. Z Gesamte Hyg 35(2):96-97. OTHER No references were identified for this sampling matrix for this agent. SOIL MATRIX Poole SK, Poole CF, Chromatographic models for the sorption of neutral organic compounds by soil from water and air. J Chromatogr A 845(1-2):381-400. SURFACES Almuzara C, Cos O, Baeza M, Gabriel D, Valero F, Methanol determination in Pichia pastoris cultures by flow injection analysis. Biotechnol Lett 24(5):413-417. WATER Blanco M, Coello J, Iturriaga H, Maspoch S, Porcel M, Simultaneous enzymatic spectrophotometric determination of ethanol and methanol by use of artificial neural networks for calibration. Anal Chim Acta 398(1):83-92.Martinezsegura G, Rivera MI, Garcia LA, Methanol analysis by gas-chromatography–comparative-study using 3 different columns. J Agric Univ Puerto Rico 69(2):135-144. Pettersson J, Roeraade J, Quantitative accuracy in the gas chromatographic analysis of solvent mixtures. J Chromatogr A 985(1-2):21-27. Wilson LA, Ding JH, Woods AE, Gas-chromatographic determination and pattern-recognition analysis of methanol and fusel oil concentrations in whiskeys. J Assoc Off Anal Chem 74(2):248-256.
TIME COURSE : Adverse health effects from methanol poisoning may not become apparent until after an asymptomatic period of 1 to 72 hours. EFFECTS OF SHORT-TERM (LESS THAN 8-HOURS) EXPOSURE : Methanol’s toxicity is due to its metabolic products. The by-products of methanol metabolism cause an accumulation of acid in the blood (metabolic acidosis), blindness, and death. Initial adverse health effects due to methanol poisoning include drowsiness, a reduced level of consciousness (CNS depression), confusion, headache, dizziness, and the inability to coordinate muscle movement (ataxia). Other adverse health effects may include nausea, vomiting (emesis), and heart and respiratory (cardiopulmonary) failure. Prognosis is poor in patient/victims with coma or seizure and severe metabolic acidosis (pH <7). Early on after methanol exposure, there may be a relative absence of adverse health effects. This does not imply insignificant toxicity. Methanol toxicity worsens as the degree of metabolic acidosis increases, and thus, becomes more severe as the time between exposure and treatment increases. EYE EXPOSURE :
Irritation, redness, and pain.
INGESTION EXPOSURE :
Ingestion of methanol may cause a wide range of adverse health effects:
Neurological: headache, dizziness, agitation, acute mania, amnesia, decreased level of consciousness including coma, and seizure. Gastrointestinal: Nausea, vomiting, lack of an appetite (anorexia), severe abdominal pain, gastrointestinal bleeding (hemorrhage), diarrhea, liver function abnormalities, and inflammation of the pancreas (pancreatitis). Ophthalmologic: visual disturbances, blurred vision, sensitivity to light (photophobia), visual hallucinations (misty vision, skin over the eyes, snowstorm, dancing spots, flashes), partial to total loss of vision, and rarely eye pain. Visual examination may reveal abnormal findings. Fixed dilated pupils are a sign of severe exposure to methanol. Other: Electrolyte imbalances. Kidney failure, blood in the urine (hematuria), and muscle death at the cellular level (rhabdomyolysis) have been reported in severe poisonings. Fatal cases often present with fast heart rate (tachycardia) or slow heart rate (bradycardia) and an increased rate of respiration. Low blood pressure (hypotension) and respiratory arrest occur when death is imminent.
INHALATION EXPOSURE : SKIN EXPOSURE :
Irritation. See Ingestion Exposure.
INTRODUCTION : The purpose of decontamination is to make an individual and/or their equipment safe by physically removing toxic substances quickly and effectively. Care should be taken during decontamination, because absorbed agent can be released from clothing and skin as a gas. Your Incident Commander will provide you with decontaminants specific for the agent released or the agent believed to have been released. DECONTAMINATION CORRIDOR : The following are recommendations to protect the first responders from the release area:
Position the decontamination corridor upwind and uphill of the hot zone. The warm zone should include two decontamination corridors. One decontamination corridor is used to enter the warm zone and the other for exiting the warm zone into the cold zone. The decontamination zone for exiting should be upwind and uphill from the zone used to enter. Decontamination area workers should wear appropriate PPE. See the PPE section of this card for detailed information. A solution of detergent and water (which should have a pH value of at least 8 but should not exceed a pH value of 10.5) should be available for use in decontamination procedures. Soft brushes should be available to remove contamination from the PPE. Labeled, durable 6-mil polyethylene bags should be available for disposal of contaminated PPE.
INDIVIDUAL DECONTAMINATION : The following methods can be used to decontaminate an individual:
Decontamination of First Responder:
Begin washing PPE of the first responder using soap and water solution and a soft brush. Always move in a downward motion (from head to toe). Make sure to get into all areas, especially folds in the clothing. Wash and rinse (using cold or warm water) until the contaminant is thoroughly removed. Remove PPE by rolling downward (from head to toe) and avoid pulling PPE off over the head. Remove the SCBA after other PPE has been removed. Place all PPE in labeled durable 6-mil polyethylene bags.
Decontamination of Patient/Victim:
Remove the patient/victim from the contaminated area and into the decontamination corridor. Remove all clothing (at least down to their undergarments) and place the clothing in a labeled durable 6-mil polyethylene bag. Thoroughly wash and rinse (using cold or warm water) the contaminated skin of the patient/victim using a soap and water solution. Be careful not to break the patient/victim’s skin during the decontamination process, and cover all open wounds. Cover the patient/victim to prevent shock and loss of body heat. Move the patient/victim to an area where emergency medical treatment can be provided.
GENERAL INFORMATION : Initial treatment is primarily supportive of respiratory and cardiovascular function. The goal of treatment is to either prevent the conversion of methanol to toxic metabolites or to rapidly remove the toxic metabolites and correct metabolic and fluid abnormalities. ANTIDOTE : Fomepizole and ethanol are effective antidotes against methanol toxicity. Fomepizole or ethanol should be administered as soon as possible once the patient/victim has been admitted to a medical care facility. See Long Term Implications: Medical Treatment for further instruction. EYE :
Immediately remove the patient/victim from the source of exposure. Immediately wash eyes with large amounts of tepid water for at least 15 minutes. Seek medical attention immediately.
INGESTION :
Immediately remove the patient/victim from the source of exposure. Ensure that the patient/victim has an unobstructed airway. Do not induce vomiting (emesis). Seek medical attention immediately.
INHALATION :
Immediately remove the patient/victim from the source of exposure. Evaluate respiratory function and pulse. Ensure that the patient/victim has an unobstructed airway. If shortness of breath occurs or breathing is difficult (dyspnea), administer oxygen. Assist ventilation as required. Always use a barrier or bag-valve-mask device. If breathing has ceased (apnea), provide artificial respiration. Seek medical attention immediately.
SKIN :
Immediately remove the patient/victim from the source of exposure. See the Decontamination section for patient/victim decontamination procedures. Seek medical attention immediately.
MEDICAL TREATMENT : Antidotes fomepizole or ethanol should be administered intravenously as soon as possible to block the conversion of methanol to formic acid and prevent acidosis. Fomepizole is preferred as its efficacy and safety have been demonstrated, and its therapeutic dose is more easily maintained. Once the patient/victim has become acidotic, administration of fomepizole or ethanol may not provide much benefit, but they may be administered at the discretion of the physician in charge. Hemodialysis is the most effective form of treatment for an acidotic patient/victim. Folinic acid (leucovorin) should also be administered intravenously to increase the rate at which formate is metabolized into less toxic chemicals. DELAYED EFFECTS OF EXPOSURE : The most common permanent adverse health effects following severe methanol poisoning are damage to or death of the nerve leading from the eye to the brain (optic neuropathy or atrophy), resulting in blindness; disease caused by damage to a particular region of the brain, resulting in difficulty walking and moving properly (Parkinsonism); damage to the brain caused by exposure to toxins, resulting in abnormal thought (encephalopathy); and damage to the peripheral nervous system. EFFECTS OF CHRONIC OR REPEATED EXPOSURE : Methanol is not suspected to be a carcinogen. Chronic or repeated exposure to methanol is suspected to be a developmental toxicity risk. It is unknown whether chronic or repeated exposure to methanol is a reproductive toxicity risk. Methanol may cause birth defects of the central nervous system in humans. Chronic poisoning from repeated exposure to methanol vapor may produce inflammation of the eye (conjunctivitis), recurrent headaches, giddiness, insomnia, stomach disturbances, and visual failure. The most noted health consequences of longer-term exposure to lower levels of methanol are a broad range of effects on the eye. Inflammatory changes and irritation of the skin (dermatitis), occurs with chronic or repeated exposure to methanol.
INCIDENT SITE :
Consult with the Incident Commander regarding the agent dispersed, dissemination method, level of PPE required, location, geographic complications (if any), and the approximate number of remains. Coordinate responsibilities and prepare to enter the scene as part of the evaluation team along with the FBI HazMat Technician, local law enforcement evidence technician, and other relevant personnel. Begin tracking remains using waterproof tags.
RECOVERY AND ON-SITE MORGUE :
Wear PPE until all remains are deemed free of contamination. Establish a preliminary (holding) morgue. Gather evidence, and place it in a clearly labeled impervious container. Hand any evidence over to the FBI. Remove and tag personal effects. Perform a thorough external evaluation and a preliminary identification check. See the Decontamination section for decontamination procedures. Decontaminate remains before they are removed from the incident site.
See Guidelines for Mass Fatality Management During Terrorist Incidents Involving Chemical Agents, U.S. Army Soldier and Biological Chemical Command (SBCCOM), November, 2001 for detailed recommendations.
NIOSH REL :
STEL (skin): 250 ppm (325 mg/m 3 ) TWA (skin): 200 ppm (260 mg/m 3 )
OSHA PEL :
TWA (8-hour): 200 ppm (260 mg/m 3 )
ACGIH TLV :
STEL (skin): 250 ppm TLV (skin): 200 ppm
NIOSH IDLH : 6,000 ppm DOE TEEL :
TEEL-0: 250 mg/m 3 TEEL-1: 694 mg/m 3 TEEL-2: 2,750 mg/m 3 TEEL-3: 9,300 mg/m 3
AIHA ERPG :
ERPG-1: 200 ppm ERPG-2: 1,000 ppm ERPG-3: 5,000 ppm
10 min | 30 min | 60 min | 4 hr | 8 hr | |
---|---|---|---|---|---|
AEGL 1 (discomfort, non-disabling) – ppm | 670 ppm | 670 ppm | 530 ppm | 340 ppm | 270 ppm |
AEGL 2 (irreversible or other serious, long-lasting effects or impaired ability to escape) – ppm | 11,000 ppm* | 4,000 ppm | 2,100 ppm | 730 ppm | 520 ppm |
AEGL 3 (life-threatening effects or death) – ppm | ** | 14,000 ppm* | 7,200 ppm* | 2,400 ppm | 1,600 ppm |
Lower Explosion Limit (LEL) = 55,000 ppm * = > 10% LEL; ** = > 50% LEL AEGL 3 – 10 min = ** 40,000 ppm For values denoted as * safety consideration against the hazard(s) of explosion(s) must be taken into account For values denoted as ** extreme safey considerations against the hazard(s) of explosion(s) must be taken into account Level of Distinct Order Awareness (LOA) = 8.9 ppm IMPORTANT NOTE: Interim AEGLs are established following review and consideration by the National Advisory Committee for AEGLs (NAC/AEGL) of public comments on Proposed AEGLs. Interim AEGLs are available for use by organizations while awaiting NRC/NAS peer review and publication of Final AEGLs. Changes to Interim values and Technical Support Documents may occur prior to publication of Final AEGL values. In some cases, revised Interim values may be posted on this Web site, but the revised Interim Technical Support Document for the chemical may be subject to change. (Further information is available through ).
Which alcohol has the most methanol?
Things you should know: Difference between ethanol and methanol Don your white lab coats, dust off your beakers and slip on your protective goggles – it’s time to get scientific. By Adam Devermann. We recently tackled the question of ; also sharing how some distilleries are now producing ethanol-based sanitisers.
In this piece, we’re pulling you deeper into the science of booze with an article from Issue 23 of DRiNK Magazine. Ethanol, or ethyl alcohol, is the technical name for what most of us just refer to as “alcohol.” It is the most commonly used member of the alcohol family (chemicals that end in “-ol”), and is volatile, flammable and maintains a strong odour.
For you geeks, the molecular formula is CH 3 CH 2 OH. Beer, wine and spirits all contain varying amounts of ethanol intended for human consumption, though the most common use for ethanol is actually as a fuel additive. Other industrial uses include the manufacture of solvents, plastics, drugs, perfumes, anti-bacterial gels and cosmetics.
Methanol, meanwhile, is the simplest type of alcohol and is also referred to as “wood alcohol.” The molecular formula is CH 3 OH. Chemists describe it as light, volatile, flammable, slightly sweet and having a pungent odour. I would not recommend tasting it or even getting it near your skin because in quantities as small as 10ml it can cause blindness and just under 100ml will kill you.
Despite the danger, methanol is a highly traded commodity. Up to 40 percent is used to make other chemicals such as formaldehyde, which in turn is used to produce numerous products such as plastics, paints, textiles, dyes, adhesives, anti-freeze and fuel.
- It is even present in trace amounts in the atmosphere – and in all our beloved spirits.
- Perhaps the most well-known case is tequila.
- Methanol is a natural by-product of making tequila, and according to Mexican law it must actually contain methanol.
- The minimum amount required is 0.3 g/L (grams/Litre) and the maximum allowed is 3g/L.
One hundred percent agave tequilas generally have the highest levels of methanol while mixtos have significantly less. This can cause issues with tequila arriving in China as the government has set the bar for the maximum amount of methanol allowed in spirits not made from grain at 2g/L. Agave plant The methanol in tequila is present for a number of reasons including the strains of yeast used, though the main culprit is the natural pectin found in the agave plant. But despite the Chinese regulations, methanol is not unique to tequila.
- Methanol can be found in numerous other beverages.
- One example is wine, especially red, which generally contains low levels of methanol, though they can contain as much as tequila.
- The International Organization of Vine and Wine (OIV) sets the same maximum methanol standard of 3g/L as the Mexican government.
Brandy and beer also contain low levels of methanol; even orange juice and coffee are offenders – although even extreme consumers of orange juice (2 litres per day) will have less than the maximum advised daily intake of 600mg of methanol, as set by the UK Department of Health.
Methanol does however have a darker side. Since it is less expensive than ethanol and readily available, corrupt manufacturers of fake spirits will often use it as an additive, often in fatal amounts. Cases of methanol poisonings due to dubious bottles happen around the world. In September 2012, as many as 25 people died in the Czech Republic, with many more being admitted to hospitals after drinking methanol-laced spirits.
The Czech government enacted a prohibition on all spirits over 20% abv. In December 2011 as many as 40 people died and 150 people were hospitalized after consuming fake spirits in West Bengal, India. Another 17 died during New Year celebrations that same year in Southern India.
In China, fakes are also rampant, but if you happen to ingest methanol, fortunately there is an antidote. Ironically, the antidote is ethanol. Ethanol acts to block the toxic characteristics of methanol, allowing it to pass safely through your system. Unfortunately, it is not as easy as drinking a glass of whiskey for a cure – but considering that both methanol and ethanol are produced when making spirits, this might be nature’s attempt at equilibrium.
Tags : : Things you should know: Difference between ethanol and methanol
How do you avoid methanol in distillation?
Home Distiller This hobby is fun & enjoyable, but it is not tiddlywinks. Be safe! Moderator: Novice Posts: Joined: Mon Aug 13, 2018 12:43 pm by » Tue Aug 14, 2018 9:25 am Hey guys. Hoping to get some incite from your experiences. Is there a simple test for distinguishing if your drink has ethanol or methanol? I thought it was the flame test but apparently youtube says its not accurate.
- Distiller Posts: Joined: Tue Feb 09, 2016 4:29 pm by » Tue Aug 14, 2018 9:39 am There is a test with several, at least 5, reagents.
- And of course gas chromatography.
- But when you use fruits, your drinks have both methanol and ethanol.
- When you use grains or sugar there should be hardly any methanol present.
But all in all: when you ferment carefully, you do not really have to worry about methanol. Novice Posts: Joined: Mon Aug 13, 2018 12:43 pm by » Tue Aug 14, 2018 10:29 am anything simple for someone who doesn’t have reagents? Any household items can work? Master of Distillation Posts: Joined: Tue Mar 18, 2014 7:01 am Location: where the buffalo roam, and the deer & antelope play by » Tue Aug 14, 2018 11:24 am kirr45 wrote: anything simple for someone who doesn’t have reagents? Any household items can work? Read Kareltje’s response again.
There is no SIMPLE tool to determine the methanol vs ethanol in a spirit. Also, methanol forms an azeotrope with ethanol, so it is not easily separated without expensive lab equipment and significant lab experience. The BEST way to avoid methanol in a distillate is to simply “don’t make it”, That is, pay attention to the fermentation processes and avoid ingredients which will contain higher amounts of methanol, such as fruit stems and sticks, etc.
Grains and sugar ferments won’t have methanol (not measureable amounts anyway). kirr45, you’ve got some homework to do.I’d suggest starting with Cranky’s Spoon Feed thread hotlinked in my signature. It’ll answer most if not all of your upcoming questions (before you even have to ask). Posts: Joined: Sun Oct 16, 2016 3:29 pm Location: At the edge of the Wild Wood by » Tue Aug 14, 2018 1:07 pm kirr45 wrote: anything simple for someone who doesn’t have reagents? Any household items can work? Mate you’ve been reading Propaganda ! No normal recipe is going to make Methanol and you CANNOT “make Methanol” by distilling anything you have fermented.
Areltje – and Still Stirrin’ done told you good ! Master of Distillation Posts: Joined: Wed Oct 25, 2006 3:19 am by » Tue Aug 14, 2018 1:17 pm Methanol is smeared threw out. Heads you can get you a 1/2 pint jar an put some in. Bend over it an put you eye over it. Do it sting. That’s how the ole boys was taught.
Course that was then an not now. Mite not be so today So I’m tole Novice Posts: Joined: Mon Aug 13, 2018 12:43 pm by » Tue Aug 14, 2018 9:51 pm Then what about all those cases of people going blind, etc. you hear about? I guess I’m just so new at this that I wouldn’t even wanna risk,00001% becoming blind.
Do you know how many movies I haven’t seen yet? Not to mention my favorite.Dumb & Dumber. Site Mod Posts: Joined: Thu Mar 17, 2011 2:13 am Location: Northern NSW Australia by » Tue Aug 14, 2018 10:05 pm kirr45 wrote: Then what about all those cases of people going blind, etc. you hear about? Could you supply more information or links to some of these cases you speak of? Don’t believe everything you read or are told about methanol poisoning by the media and or governments.
Let me ask you a question, something for you to think about. The only country where it is legal that I know of to distill is New Zealand. I would presume that as it is legal there,many New Zealanders would distill on a daily basis and even more of the local people would drink the results of that distilling.
We can also probably assume that a lot of those people are not members of this or other forums and some of them might distill in a fairly rough and ready way. Some of them may even be making booze that has a little higher than average methanol content. Now my question.How many cases of methanol poisoning do you hear reported by the media in New Zealand each year? Without delving to far into it I reckon the answer will be close to Zero.
By the way an interesting fact for you,the antidote for methanol poisoning is Ethanol. Last edited by on Tue Aug 14, 2018 11:53 pm, edited 1 time in total. Site Donor Posts: Joined: Tue Dec 05, 2017 4:08 pm by » Tue Aug 14, 2018 10:31 pm kirr45 wrote: Then what about all those cases of people going blind, etc. you hear about? Back in U.S. prohibition days, unscrupulous people out for a buck were making drink out of all kinds of things like embalming fluid and industrial alcohol without concern about poisoning people.
As a result of drinking something like wood alcohol, the extremely high amount of methanol would metabolize into formic acid in the body, attacking the fragile optic nerve causing blindness or even death. You won’t even approach that level of methanol when administering this hobby. And you know what the cure for methanol poisoning is even if you did? Ethanol,
The very drink you’re distilling. and you will eliminate more nasty alcohols than what you buy at the store. Put away your fears. No one here has gone blind, nor will they ever from this hobby. Last edited by on Tue Aug 14, 2018 10:37 pm, edited 2 times in total. Posts: Joined: Sat Nov 05, 2016 6:24 am Location: NW Lake Gitchegumee by » Tue Aug 14, 2018 10:35 pm Saltbush Bill wrote: Could you supply more information or links to some of these cases you speak of? I’ve seen some of the stuff on youtube I watched to and was just baffled at the whole ordeal ” onclick=”window.open(this.href);return false;” rel=”nofollow I don’t know how a man drank 2 liters of 160 proof liquor but apparently it happened and it stoked the anti distilling propaganda Rye whisky rye whisky oh dont let me down Gunna have me a drink then gambol around Here’s some fiddle music Site Donor Posts: Joined: Sun Oct 16, 2016 3:29 pm Location: At the edge of the Wild Wood by » Wed Aug 15, 2018 5:25 am Fiddleford wrote: Saltbush Bill wrote: Could you supply more information or links to some of these cases you speak of? I’ve seen some of the stuff on youtube I watched to and was just baffled at the whole ordeal ” onclick=”window.open(this.href);return false;” rel=”nofollow I don’t know how a man drank 2 liters of 160 proof liquor but apparently it happened and it stoked the anti distilling propaganda Thanks for that Fiddleford What a load of old bollox ! “Ethanol spontaneously combustible” We know that “moonshine” made from Corn and sugar – WILL be Low methanol Drank 2 litres at 80%abv then felt unwell ! He was a youngish man I think – (80 kg body weight) – 2 litres @ 80% is almost certain to have killed him ! He would certainly have been comatose ! I certainly could not drink more than 1/2 litre at than strength and remain coherent ! – That would put me to sleep and I’m half as heavy again with a long history of “drinking” No idea what that guy’s motive is but he says heating the wash to 173 degrees will harvest the ethanol and the methanol.
- We know that is comletely untrue as well.
- To the OP – Read these threads We’ve been here so many times it just gets boring ! Novice Posts: Joined: Sun Mar 26, 2017 11:55 am by » Wed Aug 15, 2018 1:18 pm Why couldn’t you bring a pot of water up to temperature (around 170) and sit your jar in that? Thats above the Methanol boiling temp and below the ethanol boiling temp.
If there were any in it, it shouldn’t be for long. Master of Distillation Posts: Joined: Tue Mar 18, 2014 7:01 am Location: where the buffalo roam, and the deer & antelope play by » Wed Aug 15, 2018 2:41 pm BamaBill wrote: Why couldn’t you bring a pot of water up to temperature (around 170) and sit your jar in that? Thats above the Methanol boiling temp and below the ethanol boiling temp.
If there were any in it, it shouldn’t be for long. Try it. The methanol won’t boil because it is in solution with ethanol and some water, among other constituents like esters, oils, aldehydes, etc. As a result, the boiling point (of the mixture) is higher than the boiling point of pure methanol. This is a fundamental rule of distillation.even if you don’t like it.
It’s the physics of distillation. Review Raoult’s Law to gain more knowledge. ss Site Donor Posts: Joined: Sun Oct 16, 2016 3:29 pm Location: At the edge of the Wild Wood by » Wed Aug 15, 2018 2:57 pm BamaBill wrote: Why couldn’t you bring a pot of water up to temperature (around 170) and sit your jar in that? Thats above the Methanol boiling temp and below the ethanol boiling temp. Posts: Joined: Sun Oct 16, 2016 3:29 pm Location: At the edge of the Wild Wood by » Wed Aug 15, 2018 3:00 pm Pikey wrote: BamaBill wrote: Why couldn’t you bring a pot of water up to temperature (around 170) and sit your jar in that? Thats above the Methanol boiling temp and below the ethanol boiling temp.
If there were any in it, it shouldn’t be for long. You’ve been here a year and a half – That is sooooo obvious – why do you think nobody else has suggested it ? apart from dumb you-tube vids ! As SS says “try it ” retired Posts: Joined: Wed Mar 14, 2012 9:04 am Location: Central Highlands Vic. Australia by » Wed Aug 15, 2018 6:34 pm goose eye wrote: Methanol is smeared threw out.
Heads you can get you a 1/2 pint jar an put some in. Bend over it an put you eye over it. Do it sting. That’s how the ole boys was taught. Course that was then an not now. Mite not be so todaySo I’m tole Ole eyes much divergent of new ones ? @ kirr45 you been reading the government’s fairytales, maybe google destructive distillation.
Making methanol in any unsafe amount (with ethanol) is darn hard and with the advise within this forum practically impossible, some trace amounts of methanol is in virtually everything, even your own gut produces some. In almost every case of methanol poisoning something was added either out out greed or ignorance and guess what the most common cure is? Ethanol, ain’t that something Those who would give up essential Liberty, to purchase a little temporary Safety, deserve neither Liberty nor Safety.
Benjamin Franklin Rumrunner Posts: Joined: Tue Aug 22, 2017 1:12 pm Location: New Mexico USA by » Wed Aug 15, 2018 7:43 pm Even commercial booze has some methanol in it, just not very much. The stuff we make has some in it but probably even less than commercial since the hobbists tend to be more particular about cuts.
How much methanol is toxic?
Toxicokinetics – A potentially lethal dose of methanol is approximately 30 to 240 mL or 1 gram per kilogram. Permanent visual damage may occur with minimum ingestion of 30 mL of methanol. The parent compound, methanol, accounts for the increased osmolality.
- Unlike most other alcohols, methanol itself is not inebriating, and this may be related to its lower molecular weight.
- Formic acid is the primary toxic metabolite that accounts for the associated anion gap metabolic acidosis and end-organ damage.
- Therefore, as methanol is metabolized, the osmolar gap decreases, and the anion gap increases.
The development of an anion gap metabolic acidosis associated with formate accumulation is multifactorial, due to the accumulation of organic acids that are not easily eliminated (for example, formic acid and formate), and the disruption of oxidative phosphorylation due to formate’s inhibition of cytochrome oxidase.
- Formate’s hindrance of mitochondrial respiration can also cause a degree of lactatemia, which can enhance formate’s ability to cross the blood-brain barrier as formic acid.
- Lactate is also elevated secondary to enhanced shunting of pyruvate to lactate from the increased NADH/NAD ratio associated with alcohol metabolism.
End organ damage and retinal toxicity are primarily due to formic acid’s oxidative stress. Also reported is parkinsonian-like symptomatology associated with observed basal ganglia lesions, particularly in the putamen and globus pallidus. This is potentially due to the parent compound, methanol.
Can methanol be produced naturally?
Natural sources – Methanol is produced from a wide range of microbiological processes (from decaying organic matter), heated wood and sites of geothermal activity, such as hot springs and volcanoes.
Can you cure methanol poisoning with alcohol?
Abstract – Seven patients with methanol poisoning were treated with ethanol, hemodialysis and supportive measures. The interval between ingestion and initiation of ethanol therapy varied from 3 to 67 hours and from ingestion to dialysis from 9 to 93 hours.
Can you accidentally brew methanol?
Every few months, reports circulate of large numbers of people falling ill after a wedding or other gathering. The illnesses – often deaths too – are blamed on bad alcohol; cheaply made homebrew which contains potentially lethal levels of methanol. Big outbreaks of methanol poisoning make the news, but the reality is that every day around the world people gamble on a bottle of cheap booze.
- And experts say that in many places, if something does go wrong, medical staff simply don’t have the awareness to stave off critical injury or death.
- In a slum district of Jakarta, Indonesia, Kardi recalls his years of drinking bootleg alcohol.
- It was his “energiser” for work as a motorbike mechanic, he says.
“I don’t mean to brag but I had money. I drank every day.” But one day, one of the regular customers at his workshop in Jakarta turned up with some cheap bootleg, known in Indonesia as oplasan. It cost about 7,500 rupiah a bottle (£0.42; $0.53). “I drank eight of them”, says Kardi.
- I could work on two to three motorcycles but I ended up not touching the fourth.
- I kept drinking.
- Next thing I knew it was 2am.” Overnight, it became clear he was suffering from more than a hangover.
- Ardi woke up in the night with crippling stomach pains.
- It was like.
- This,” he says, crunching a plastic bottle in his hands.
He tried to make himself sick, but “it was all black” and he eventually passed out. Image caption, Kardi says he feels lucky that he survived when some of his friends didn’t A family member took him to a hospital, but the staff refused to treat him, he says, because they were Muslim and he had been drinking – not an uncommon occurrence.
A second hospital was also about to turn him away but, by chance, he had fixed the motorbike of one of the doctors on duty. This doctor agreed to treat him and immediately diagnosed methanol poisoning. Methanol is produced during the brewing process and concentrated by distillation. Commercial manufacturers will reduce it to levels which are safe for human consumption.
However, unscrupulous backyard brewers or others in the supply chain may sometimes add industrially produced methanol, to make it go further and increase profits. It is also possible for high levels of methanol to be produced by contaminating microbes during traditional ethanol fermentation.
- Overnight, Kardi’s body had finished processing the ethanol from the oplosan.
- Next, it started on the methanol, metabolising it into highly toxic formic acid.
- His body gradually became more and more acidic, giving him chest pains, gastric pain, headaches and blurred vision.
- The doctor was able to save his life, but Kardi’s injuries were life-changing.
He suffered temporary full-body paralysis and full movement was never restored, His eyes had been severely damaged – over the following years he lost his sight almost completely. His wife left him and he is now dependent on his community to get by. Kardi’s cloudy eyes are fixed in the middle distance as he says that in some ways he was lucky.
How does alcohol get tainted with methanol?
Methanol poisonings by direct consumption occur when unscrupulous enterprises or individuals add industrial methanol into legitimate alcoholic beverages. The poisonings can also be caused through improper brewing of homemade alcohol, which produces methanol instead of ethanol.
Does fermenting fruit make methanol?
Abstract – Methanol is a natural ingredient with major occurrence in fruit spirits, such as apple, pear, plum or cherry spirits, but also in spirits made from coffee pulp. The compound is formed during fermentation and the following mash storage by enzymatic hydrolysis of naturally present pectins.
Methanol is toxic above certain threshold levels and legal limits have been set in most jurisdictions. Therefore, the methanol content needs to be mitigated and its level must be controlled. This article will review the several factors that influence the methanol content including the pH value of the mash, the addition of various yeast and enzyme preparations, fermentation temperature, mash storage, and most importantly the raw material quality and hygiene.
From all these mitigation possibilities, lowering the pH value and the use of cultured yeasts when mashing fruit substances is already common as best practice today. Also a controlled yeast fermentation at acidic pH facilitates not only reduced methanol formation, but ultimately also leads to quality benefits of the distillate.
How is methanol typically removed from moonshine?
With the dangers of methanol in mind, it would seem downright foolish to consume any spirit, let alone moonshine. However, there is a way to remove methanol from moonshine. In order to remove the harmful byproducts from your shine you need to perform what is called cuts and fractions during distillation.
How can you reduce the exposure to methanol?
How can I reduce my exposure to methanol? Wear appropriate protective gear when using paints, adhesives, and varnishes. Washing your hands after using these items also reduces exposure. If you work with methanol, follow the protection requirements in your workplace.
How do I make sure moonshine is safe to drink?
Is It Safe to Drink Moonshine? With the drinking culture we have developed in the past few decades, liquor stores and bars alike have acquired a wide variety of specialty wines, beers, and spirits. Among the fastest-growing crazes is the resurgence of moonshine,
- This formerly hush-hush, home-distilled drink of backwoods Appalachia is now legit! Its sale is being regulated by the US government in some states.
- So what exactly is moonshine? How come it is winning the hearts of several craft distillers despite being one of the most illicit drinks in the US? Is it really safe to drink moonshine? What is Moonshine? Moonshine, more popularly known as “hooch” or “homebrew” is made from a mash of corn, sugar, yeast, and water.
Unlike whiskey and bourbon, moonshine is not aged. It has a very high alcohol content, sometimes peaking at 190 proof. Traditionally, it is being produced in a homemade moonshine still and bottled in a mason jar. It was first introduced in the US in the 18 th century by Scottish and Irish immigrants.
- It quickly became a mainstay of Southern culture.
- As its popularity crested, the government became interested in taxation.
- People started making it in the middle of the night, under the light of the moon, hence its name.
- This is to hide the smoke that would run off the boiling liquor from local law enforcers.
Today, moonshine is widely available in some states and online for purchase. Why Do People Love Drinking Moonshine? While most people drink it straight from the jar, moonshine is excellent for mixing cocktails! It is basically whiskey, so you can combine it with Coke, ginger ale, lemonade, iced tea, and more! You can also infuse fresh apples, pineapples, peaches, blackberry, blueberry, cinnamon, or tangerine juice to this drink.
- The possibilities are endless! Is It Safe To Drink Moonshine? You probably have read stories about the dangers of moonshine and other illegally distilled drinks.
- Illegal moonshine remains to be dangerous because it is brewed in makeshift stills.
- The distillation process produces alcohol vapors, which are highly flammable.
The threat of explosion is too high, hence moonshine stills are better outdoors. Although most of the stills in operation today are all-copper variety, plenty of old vehicles are still around. These old handmade stills use lead-based solder to make connections.
Unfortunately, lead can leach out of the radiators and connections. Lead is toxic to the body. Lead poisoning can cause memory loss, brain swelling, paralysis, and even death. In larger batches of distilled moonshine, tainting with methanol can occur. Methanol vaporizes at a lower temperature than ethanol, so it is the first liquid to be produced during the distillation process.
The larger the batch, the more ethanol. Methanol is highly poisonous. When metabolized in the body, it can cause serious side effects. With just a few ml, the dangerous potential of methanol can be undetectable. You will just get a hangover. However, 10 ml or more of methanol, even split up among drinks, can permanently damage your optic nerve and cause partial or complete blindness.
- The lethal dose is 30 ml.
- How Can You Tell Which Moonshine Is Safe To Drink? One way to test the purity of a moonshine liquor is to pour some in a metal spoon and set it on fire.
- If it burns with a blue flame, it is more likely safe to drink.
- If it produces a yellow or red flame, it is an indication of the presence of lead.
However, this spoon test does not detect the presence of methanol, which burns a colorless flame. With hundreds and thousands of moonshine being produced on a regular basis, chances are some of it will be tainted. When made properly, moonshine is simply a strong alcohol with a very hard taste or “kick” because it has not been aged.
What can neutralize methanol?
Methanol: Systemic Agent
CAS #: 67-56-1 RTECS #: PC1400000 UN #: 1230 (Guide 131)
Common Names:
Carbinol Methyl alcohol Wood alcohol
APPEARANCE : Colorless watery liquid. DESCRIPTION : Methanol is a toxic alcohol that is used industrially as a solvent, pesticide, and alternative fuel source. It also occurs naturally in humans, animals, and plants. Foods such as fresh fruits and vegetables, fruit juices, fermented beverages, and diet soft drinks containing aspartame are the primary sources of methanol in the human body. Most methanol poisonings occur as a result of drinking beverages contaminated with methanol or from drinking methanol-containing products. In the industrial setting, inhalation of high concentrations of methanol vapor and absorption of methanol through the skin are as effective as the oral route in producing toxic effects. The characteristic pungent (alcohol) odor of methanol does not provide sufficient warning of low levels of exposure. METHODS OF DISSEMINATION :
Indoor Air: Methanol can be released into indoor air as a liquid spray (aerosol). Water: Methanol can be used to contaminate water. Food: Methanol may be used to contaminate food. Outdoor Air: Methanol can be released into outdoor air as a liquid spray (aerosol). Agricultural: If methanol is released into the air as a liquid spray (aerosol), it has the potential to contaminate agricultural products.
ROUTES OF EXPOSURE : Methanol can be absorbed into the body by inhalation, ingestion, skin contact, or eye contact. Ingestion is an important route of exposure.
GENERAL INFORMATION : First Responders should use a NIOSH-certified Chemical, Biological, Radiological, Nuclear (CBRN) Self Contained Breathing Apparatus (SCBA) with a Level A protective suit when entering an area with an unknown contaminant or when entering an area where the concentration of the contaminant is unknown. Level A protection should be used until monitoring results confirm the contaminant and the concentration of the contaminant. NOTE: Safe use of protective clothing and equipment requires specific skills developed through training and experience. LEVEL A: (RED ZONE) : Select when the greatest level of skin, respiratory, and eye protection is required. This is the maximum protection for workers in danger of exposure to unknown chemical hazards or levels above the IDLH or greater than the AEGL-2.
A NIOSH-certified CBRN full-face-piece SCBA operated in a pressure-demand mode or a pressure-demand supplied air hose respirator with an auxiliary escape bottle. A Totally-Encapsulating Chemical Protective (TECP) suit that provides protection against CBRN agents. Chemical-resistant gloves (outer). Chemical-resistant gloves (inner). Chemical-resistant boots with a steel toe and shank. Coveralls, long underwear, and a hard hat worn under the TECP suit are optional items.
LEVEL B: (RED ZONE) : Select when the highest level of respiratory protection is necessary but a lesser level of skin protection is required. This is the minimum protection for workers in danger of exposure to unknown chemical hazards or levels above the IDLH or greater than AEGL-2.
A NIOSH-certified CBRN full-face-piece SCBA operated in a pressure-demand mode or a pressure-demand supplied air hose respirator with an auxiliary escape bottle. A hooded chemical-resistant suit that provides protection against CBRN agents. Chemical-resistant gloves (outer). Chemical-resistant gloves (inner). Chemical-resistant boots with a steel toe and shank. Coveralls, long underwear, a hard hat worn under the chemical-resistant suit, and chemical-resistant disposable boot-covers worn over the chemical-resistant suit are optional items.
LEVEL C: (YELLOW ZONE) : Select when the contaminant and concentration of the contaminant are known and the respiratory protection criteria factors for using Air Purifying Respirators (APR) or Powered Air Purifying Respirators (PAPR) are met. This level is appropriate when decontaminating patient/victims.
A NIOSH-certified CBRN tight-fitting APR with a canister-type gas mask or CBRN PAPR for air levels greater than AEGL-2. A NIOSH-certified CBRN PAPR with a loose-fitting face-piece, hood, or helmet and a filter or a combination organic vapor, acid gas, and particulate cartridge/filter combination or a continuous flow respirator for air levels greater than AEGL-1. A hooded chemical-resistant suit that provides protection against CBRN agents. Chemical-resistant gloves (outer). Chemical-resistant gloves (inner). Chemical-resistant boots with a steel toe and shank. Escape mask, face shield, coveralls, long underwear, a hard hat worn under the chemical-resistant suit, and chemical-resistant disposable boot-covers worn over the chemical-resistant suit are optional items.
LEVEL D: (GREEN ZONE) : Select when the contaminant and concentration of the contaminant are known and the concentration is below the appropriate occupational exposure limit or less than AEGL-1 for the stated duration times.
Limited to coveralls or other work clothes, boots, and gloves.
CHEMICAL DANGERS :
Methanol reacts violently with strong oxidants, causing a fire and explosion hazard.
EXPLOSION HAZARDS :
Mixtures of methanol vapor and air are explosive. Lower explosive (flammable) limit in air (LEL), 6.0%; upper explosive (flammable) limit in air (UEL), 36%. Agent presents a vapor explosion and poison (toxic) hazard indoors, outdoors, or in sewers. Run-off to sewers may create an explosion hazard. Containers may explode when heated.
FIRE FIGHTING INFORMATION :
Methanol is highly flammable. The agent will be easily ignited by heat, sparks, or flames. Fire will produce irritating, corrosive, and/or toxic gases. Vapors may travel to the source of ignition and flash back. Run-off to sewers may create a fire hazard. Caution: The agent has a very low flash point. Use of water spray when fighting fires may be inefficient. For small fires, use dry chemical, carbon dioxide, water spray, or alcohol-resistant foam. For large fires, use water spray, fog, or alcohol-resistant foam. Move containers from the fire area if it is possible to do so without risk to personnel. Dike fire control water for later disposal; do not scatter the agent. Use water spray or fog; do not use straight streams. For fire involving tanks or car/trailer loads, fight the fire from maximum distance or use unmanned hose holders or monitor nozzles. Cool containers with flooding quantities of water until well after the fire is out. Withdraw immediately in case of rising sound from venting safety devices or discoloration of tanks. Always stay away from tanks engulfed in fire. For massive fire, use unmanned hose holders or monitor nozzles; if this is impossible, withdraw from the area and let the fire burn. Run-off from fire control or dilution water may cause pollution. If the situation allows, control and properly dispose of run-off (effluent).
INITIAL ISOLATION AND PROTECTIVE ACTION DISTANCES :
If a tank, rail car, or tank truck is involved in a fire, isolate it for 0.5 mi (800 m) in all directions; also consider initial evacuation for 0.5 mi (800 m) in all directions. This agent is not included in the DOT ERG 2004 Table of Initial Isolation and Protective Action Distances. In the DOT ERG 2004 orange-bordered section of the guidebook, there are public safety recommendations to isolate a methanol (Guide 131) spill or leak area immediately for at least 150 ft (50 m) in all directions.
PHYSICAL DANGERS :
Methanol vapors may be heavier than air. They will spread along the ground and collect and stay in poorly-ventilated, low-lying, or confined areas (e.g., sewers, basements, and tanks). Hazardous concentrations may develop quickly in enclosed, poorly-ventilated, or low-lying areas. Keep out of these areas. Stay upwind. Liquid agent is lighter than water.
NFPA 704 Signal :
Health: 1 Flammability: 3 Reactivity: 0 Special:
SAMPLING AND ANALYSIS :
OSHA: 91 NIOSH: 2000, 3800
ADDITIONAL SAMPLING AND ANALYSIS INFORMATION : References are provided for the convenience of the reader and do not imply endorsement by NIOSH.
AIR MATRIX Allen TM, Falconer TM, Cisper ME, Borgerding AJ, Wilkerson CW Jr., Real-time analysis of methanol in air and water by membrane introduction mass spectrometry. Anal Chem 73(20):4830-4835.De Paula PP, Santos E, De Freitas FT, De Andrade JB, Determination of methanol and ethanol by gas chromatography following air sampling onto florisil cartridges and their concentrations at urban sites in the three largest cities in Brazil. Talanta 49(2):245-252. Leibrock E, Slemr J, Method for measurement of volatile oxygenated hydrocarbons in ambient air. Atmos Environ 31(20):3329-3339. Marley NA, Gaffney JS, A comparison of flame ionization and ozone chemiluminescence for the determination of atmospheric hydrocarbons. Atmos Environ 32(8):1435-1444. NIOSH, NMAM Method 2000 Methanol. In: NIOSH Manual of analytical methods.4th ed. Cincinnati, OH: U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, DHHS (NIOSH) Publication 94-113. OSHA, Methyl Alcohol Method 91. Salt Lake City, UT.U.S. Department of Labor, Organic Methods Evaluation Branch, OSHA Salt Lake Technical Center. Qin T, Xu X, Polak T, Pacakova V, Stulik K, Jech L, A simple method for the trace determination of methanol, ethanol, acetone, and pentane in human breath and in the ambient air by preconcentration on solid sorbents followed by gas chromatography. Talanta 44(9):1683-1690. Reichert J, Coerdt W, Ache HJ, Development of a surface acoustic wave sensor array for the detection of methanol in fuel vapours. Sens Actuators B: Chem 13(1-3):293-296. Tyras H, Spectrophotometric determination of methyl alcohol in the atmosphere. Z Gesamte Hyg 35(2):96-97. OTHER No references were identified for this sampling matrix for this agent. SOIL MATRIX Poole SK, Poole CF, Chromatographic models for the sorption of neutral organic compounds by soil from water and air. J Chromatogr A 845(1-2):381-400. SURFACES Almuzara C, Cos O, Baeza M, Gabriel D, Valero F, Methanol determination in Pichia pastoris cultures by flow injection analysis. Biotechnol Lett 24(5):413-417. WATER Blanco M, Coello J, Iturriaga H, Maspoch S, Porcel M, Simultaneous enzymatic spectrophotometric determination of ethanol and methanol by use of artificial neural networks for calibration. Anal Chim Acta 398(1):83-92.Martinezsegura G, Rivera MI, Garcia LA, Methanol analysis by gas-chromatography–comparative-study using 3 different columns. J Agric Univ Puerto Rico 69(2):135-144. Pettersson J, Roeraade J, Quantitative accuracy in the gas chromatographic analysis of solvent mixtures. J Chromatogr A 985(1-2):21-27. Wilson LA, Ding JH, Woods AE, Gas-chromatographic determination and pattern-recognition analysis of methanol and fusel oil concentrations in whiskeys. J Assoc Off Anal Chem 74(2):248-256.
TIME COURSE : Adverse health effects from methanol poisoning may not become apparent until after an asymptomatic period of 1 to 72 hours. EFFECTS OF SHORT-TERM (LESS THAN 8-HOURS) EXPOSURE : Methanol’s toxicity is due to its metabolic products. The by-products of methanol metabolism cause an accumulation of acid in the blood (metabolic acidosis), blindness, and death. Initial adverse health effects due to methanol poisoning include drowsiness, a reduced level of consciousness (CNS depression), confusion, headache, dizziness, and the inability to coordinate muscle movement (ataxia). Other adverse health effects may include nausea, vomiting (emesis), and heart and respiratory (cardiopulmonary) failure. Prognosis is poor in patient/victims with coma or seizure and severe metabolic acidosis (pH <7). Early on after methanol exposure, there may be a relative absence of adverse health effects. This does not imply insignificant toxicity. Methanol toxicity worsens as the degree of metabolic acidosis increases, and thus, becomes more severe as the time between exposure and treatment increases. EYE EXPOSURE :
Irritation, redness, and pain.
INGESTION EXPOSURE :
Ingestion of methanol may cause a wide range of adverse health effects:
Neurological: headache, dizziness, agitation, acute mania, amnesia, decreased level of consciousness including coma, and seizure. Gastrointestinal: Nausea, vomiting, lack of an appetite (anorexia), severe abdominal pain, gastrointestinal bleeding (hemorrhage), diarrhea, liver function abnormalities, and inflammation of the pancreas (pancreatitis). Ophthalmologic: visual disturbances, blurred vision, sensitivity to light (photophobia), visual hallucinations (misty vision, skin over the eyes, snowstorm, dancing spots, flashes), partial to total loss of vision, and rarely eye pain. Visual examination may reveal abnormal findings. Fixed dilated pupils are a sign of severe exposure to methanol. Other: Electrolyte imbalances. Kidney failure, blood in the urine (hematuria), and muscle death at the cellular level (rhabdomyolysis) have been reported in severe poisonings. Fatal cases often present with fast heart rate (tachycardia) or slow heart rate (bradycardia) and an increased rate of respiration. Low blood pressure (hypotension) and respiratory arrest occur when death is imminent.
INHALATION EXPOSURE : SKIN EXPOSURE :
Irritation. See Ingestion Exposure.
INTRODUCTION : The purpose of decontamination is to make an individual and/or their equipment safe by physically removing toxic substances quickly and effectively. Care should be taken during decontamination, because absorbed agent can be released from clothing and skin as a gas. Your Incident Commander will provide you with decontaminants specific for the agent released or the agent believed to have been released. DECONTAMINATION CORRIDOR : The following are recommendations to protect the first responders from the release area:
Position the decontamination corridor upwind and uphill of the hot zone. The warm zone should include two decontamination corridors. One decontamination corridor is used to enter the warm zone and the other for exiting the warm zone into the cold zone. The decontamination zone for exiting should be upwind and uphill from the zone used to enter. Decontamination area workers should wear appropriate PPE. See the PPE section of this card for detailed information. A solution of detergent and water (which should have a pH value of at least 8 but should not exceed a pH value of 10.5) should be available for use in decontamination procedures. Soft brushes should be available to remove contamination from the PPE. Labeled, durable 6-mil polyethylene bags should be available for disposal of contaminated PPE.
INDIVIDUAL DECONTAMINATION : The following methods can be used to decontaminate an individual:
Decontamination of First Responder:
Begin washing PPE of the first responder using soap and water solution and a soft brush. Always move in a downward motion (from head to toe). Make sure to get into all areas, especially folds in the clothing. Wash and rinse (using cold or warm water) until the contaminant is thoroughly removed. Remove PPE by rolling downward (from head to toe) and avoid pulling PPE off over the head. Remove the SCBA after other PPE has been removed. Place all PPE in labeled durable 6-mil polyethylene bags.
Decontamination of Patient/Victim:
Remove the patient/victim from the contaminated area and into the decontamination corridor. Remove all clothing (at least down to their undergarments) and place the clothing in a labeled durable 6-mil polyethylene bag. Thoroughly wash and rinse (using cold or warm water) the contaminated skin of the patient/victim using a soap and water solution. Be careful not to break the patient/victim’s skin during the decontamination process, and cover all open wounds. Cover the patient/victim to prevent shock and loss of body heat. Move the patient/victim to an area where emergency medical treatment can be provided.
GENERAL INFORMATION : Initial treatment is primarily supportive of respiratory and cardiovascular function. The goal of treatment is to either prevent the conversion of methanol to toxic metabolites or to rapidly remove the toxic metabolites and correct metabolic and fluid abnormalities. ANTIDOTE : Fomepizole and ethanol are effective antidotes against methanol toxicity. Fomepizole or ethanol should be administered as soon as possible once the patient/victim has been admitted to a medical care facility. See Long Term Implications: Medical Treatment for further instruction. EYE :
Immediately remove the patient/victim from the source of exposure. Immediately wash eyes with large amounts of tepid water for at least 15 minutes. Seek medical attention immediately.
INGESTION :
Immediately remove the patient/victim from the source of exposure. Ensure that the patient/victim has an unobstructed airway. Do not induce vomiting (emesis). Seek medical attention immediately.
INHALATION :
Immediately remove the patient/victim from the source of exposure. Evaluate respiratory function and pulse. Ensure that the patient/victim has an unobstructed airway. If shortness of breath occurs or breathing is difficult (dyspnea), administer oxygen. Assist ventilation as required. Always use a barrier or bag-valve-mask device. If breathing has ceased (apnea), provide artificial respiration. Seek medical attention immediately.
SKIN :
Immediately remove the patient/victim from the source of exposure. See the Decontamination section for patient/victim decontamination procedures. Seek medical attention immediately.
MEDICAL TREATMENT : Antidotes fomepizole or ethanol should be administered intravenously as soon as possible to block the conversion of methanol to formic acid and prevent acidosis. Fomepizole is preferred as its efficacy and safety have been demonstrated, and its therapeutic dose is more easily maintained. Once the patient/victim has become acidotic, administration of fomepizole or ethanol may not provide much benefit, but they may be administered at the discretion of the physician in charge. Hemodialysis is the most effective form of treatment for an acidotic patient/victim. Folinic acid (leucovorin) should also be administered intravenously to increase the rate at which formate is metabolized into less toxic chemicals. DELAYED EFFECTS OF EXPOSURE : The most common permanent adverse health effects following severe methanol poisoning are damage to or death of the nerve leading from the eye to the brain (optic neuropathy or atrophy), resulting in blindness; disease caused by damage to a particular region of the brain, resulting in difficulty walking and moving properly (Parkinsonism); damage to the brain caused by exposure to toxins, resulting in abnormal thought (encephalopathy); and damage to the peripheral nervous system. EFFECTS OF CHRONIC OR REPEATED EXPOSURE : Methanol is not suspected to be a carcinogen. Chronic or repeated exposure to methanol is suspected to be a developmental toxicity risk. It is unknown whether chronic or repeated exposure to methanol is a reproductive toxicity risk. Methanol may cause birth defects of the central nervous system in humans. Chronic poisoning from repeated exposure to methanol vapor may produce inflammation of the eye (conjunctivitis), recurrent headaches, giddiness, insomnia, stomach disturbances, and visual failure. The most noted health consequences of longer-term exposure to lower levels of methanol are a broad range of effects on the eye. Inflammatory changes and irritation of the skin (dermatitis), occurs with chronic or repeated exposure to methanol.
INCIDENT SITE :
Consult with the Incident Commander regarding the agent dispersed, dissemination method, level of PPE required, location, geographic complications (if any), and the approximate number of remains. Coordinate responsibilities and prepare to enter the scene as part of the evaluation team along with the FBI HazMat Technician, local law enforcement evidence technician, and other relevant personnel. Begin tracking remains using waterproof tags.
RECOVERY AND ON-SITE MORGUE :
Wear PPE until all remains are deemed free of contamination. Establish a preliminary (holding) morgue. Gather evidence, and place it in a clearly labeled impervious container. Hand any evidence over to the FBI. Remove and tag personal effects. Perform a thorough external evaluation and a preliminary identification check. See the Decontamination section for decontamination procedures. Decontaminate remains before they are removed from the incident site.
See Guidelines for Mass Fatality Management During Terrorist Incidents Involving Chemical Agents, U.S. Army Soldier and Biological Chemical Command (SBCCOM), November, 2001 for detailed recommendations.
NIOSH REL :
STEL (skin): 250 ppm (325 mg/m 3 ) TWA (skin): 200 ppm (260 mg/m 3 )
OSHA PEL :
TWA (8-hour): 200 ppm (260 mg/m 3 )
ACGIH TLV :
STEL (skin): 250 ppm TLV (skin): 200 ppm
NIOSH IDLH : 6,000 ppm DOE TEEL :
TEEL-0: 250 mg/m 3 TEEL-1: 694 mg/m 3 TEEL-2: 2,750 mg/m 3 TEEL-3: 9,300 mg/m 3
AIHA ERPG :
ERPG-1: 200 ppm ERPG-2: 1,000 ppm ERPG-3: 5,000 ppm
10 min | 30 min | 60 min | 4 hr | 8 hr | |
---|---|---|---|---|---|
AEGL 1 (discomfort, non-disabling) – ppm | 670 ppm | 670 ppm | 530 ppm | 340 ppm | 270 ppm |
AEGL 2 (irreversible or other serious, long-lasting effects or impaired ability to escape) – ppm | 11,000 ppm* | 4,000 ppm | 2,100 ppm | 730 ppm | 520 ppm |
AEGL 3 (life-threatening effects or death) – ppm | ** | 14,000 ppm* | 7,200 ppm* | 2,400 ppm | 1,600 ppm |
Lower Explosion Limit (LEL) = 55,000 ppm * = > 10% LEL; ** = > 50% LEL AEGL 3 – 10 min = ** 40,000 ppm For values denoted as * safety consideration against the hazard(s) of explosion(s) must be taken into account For values denoted as ** extreme safey considerations against the hazard(s) of explosion(s) must be taken into account Level of Distinct Order Awareness (LOA) = 8.9 ppm IMPORTANT NOTE: Interim AEGLs are established following review and consideration by the National Advisory Committee for AEGLs (NAC/AEGL) of public comments on Proposed AEGLs. Interim AEGLs are available for use by organizations while awaiting NRC/NAS peer review and publication of Final AEGLs. Changes to Interim values and Technical Support Documents may occur prior to publication of Final AEGL values. In some cases, revised Interim values may be posted on this Web site, but the revised Interim Technical Support Document for the chemical may be subject to change. (Further information is available through ).